Side more recently
Back more recently.
Pictures, as promised. These were taken last Friday. I went to the beach this weekend and got dark as hell, so I have to see how those pictures turn out
First one is front relaxed, this one is my attempt at a lat spread.
Big Balls, by the way how did you clean up that acne? I just started getting some on my upper back, the beach burned a lot of it off this weekend, but I don’t want it to come back.
Oh, and by the way, this was me a bit over a year ago . . . ummm . . . yeah . . . beer is good???
[quote]MapShooter wrote:
Big Balls, by the way how did you clean up that acne? I just started getting some on my upper back, the beach burned a lot of it off this weekend, but I don’t want it to come back.[/quote]
Stopped drinking milk all the time. Also a holiday in Spain with sunshine cleaned up my spots a bit.
Update on egcg:
Hepatic and Gastrointestinal Toxicities of Green Tea Polyphenols
Despite several human studies that showed no toxicity of tea polyphenol preparations and that the major adverse effects associated with consumption of high doses of tea preparations are due to gastrointestinal irritation, there have been a number of recent case reports of hepatotoxicity related to the consumption of high doses of tea-based dietary supplements (10-29 mg/kg/day po) http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00018. In nearly all cases (eight out of nine), patients presented with elevated serum alanine aminotransferase (ALT) and bilirubin levels. In two of nine cases, periportal and portal inflammation were observed. All cases resolved following cessation of supplement consumption. A causative role for the green tea preparations is suggested by the fact that reinjury was observed following rechallenge with the same preparations. The authors of these reports, however, could not conclusively rule out the involvement of potentially hepatotoxic pharmaceutical agents such as acetaminophen or other dietary supplements. One intriguing possibility is that susceptible individuals have a polymorphism in a key biotransformation pathway for the tea polyphenols, such as low activity COMT, which increases exposure to the unmetabolized parent compound. Such possibilities need to be further explored.
Laboratory studies in rodents and dogs have supported the potential toxic effects of high doses of green tea-derived preparations http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00016. Oral administration (po) of Teavigo (a green tea polyphenol preparation containing 90% EGCG) or Polyphenon E for 13 or nine weeks, respectively, to Beagle dogs resulted in dose-dependent toxicity and death http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00019. Vomiting and diarrhea were observed throughout both studies. In addition, 500 mg/kg, po, Teavigo caused proximal tubule necrosis and elevated serum bilirubin in all dogs treated. Most male dogs (2/3) had elevated serum aspartate aminotransferase (AST) levels. Female dogs (2/3), but not male dogs, had liver necrosis. Although it is likely that the catechins are responsible for the toxicity in the dogs, a role for caffeine cannot be rule out since although Teavigo is caffeine-free, Polyphenon E contains 0.7% caffeine. Oral administration of 2000 mg/kg and intragastric administration (ig) Teavigo to rats resulted in lethality in 80% of animals treated http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00019. Histological analysis revealed hemorrhagic lesions in the stomach and intestine. Intraperitoneal administration of EGCG to mice resulted in dose-dependent lethality beginning at 150 mg/kg http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00016. Lethality was associated with increases in serum ALT levels, suggesting the involvement of hepatotoxicity.
These data suggest that high doses of EGCG can induce toxicity in the liver, kidneys, and intestine. Toxicity, especially in the liver and kidney, appears to be correlated with the bioavailability of EGCG. In the rat, where bioavailability is low [absolute bioavailability (F) = 1.6%], toxicity is confined to the gastrointestinal tract following po http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00020. In the dog, where bioavailability is much higher, hepatotoxicity, nephrotoxicity, and intestinal toxicity were observed. Toxicity was greater in fasted, than in prefed, dogs http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00019. The AUCplasma (area under the curve) in the prefed dogs was 19.8 g h/mL as compared to 205 g h/mL in fasted dogs following administration of 300 mg/kg, po. Recent studies in humans have also demonstrated that fasting increases the bioavailability of EGCG http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00021. Although there have been no reports of toxicity in human volunteers enrolled in intervention studies, careful monitoring of liver and kidney function is required until the risk of toxic events associated with tea catechins is established in humans.
The doses they have the animals seem extremely high . . . If a human were given 500 mg/kilo or 2000 mg/kilo we are talking about 40 grams of tea extract! I take 2-3 grams of egcg/day and have not noticed any adverse affects, and i just had my blood panel done
The pics look good guys, not a bad transformation for you b.balls; myostain or not.
How did your lifts change as well?
But were his results from myostatin or the self-wrestling he’s been doing… That is a good thread!
[quote]big balls wrote:
Update on egcg:
Hepatic and Gastrointestinal Toxicities of Green Tea Polyphenols
Despite several human studies that showed no toxicity of tea polyphenol preparations and that the major adverse effects associated with consumption of high doses of tea preparations are due to gastrointestinal irritation, there have been a number of recent case reports of hepatotoxicity related to the consumption of high doses of tea-based dietary supplements (10-29 mg/kg/day po) http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00018. In nearly all cases (eight out of nine), patients presented with elevated serum alanine aminotransferase (ALT) and bilirubin levels. In two of nine cases, periportal and portal inflammation were observed. All cases resolved following cessation of supplement consumption. A causative role for the green tea preparations is suggested by the fact that reinjury was observed following rechallenge with the same preparations. The authors of these reports, however, could not conclusively rule out the involvement of potentially hepatotoxic pharmaceutical agents such as acetaminophen or other dietary supplements. One intriguing possibility is that susceptible individuals have a polymorphism in a key biotransformation pathway for the tea polyphenols, such as low activity COMT, which increases exposure to the unmetabolized parent compound. Such possibilities need to be further explored.
Laboratory studies in rodents and dogs have supported the potential toxic effects of high doses of green tea-derived preparations http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00016. Oral administration (po) of Teavigo (a green tea polyphenol preparation containing 90% EGCG) or Polyphenon E for 13 or nine weeks, respectively, to Beagle dogs resulted in dose-dependent toxicity and death http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00019. Vomiting and diarrhea were observed throughout both studies. In addition, 500 mg/kg, po, Teavigo caused proximal tubule necrosis and elevated serum bilirubin in all dogs treated. Most male dogs (2/3) had elevated serum aspartate aminotransferase (AST) levels. Female dogs (2/3), but not male dogs, had liver necrosis. Although it is likely that the catechins are responsible for the toxicity in the dogs, a role for caffeine cannot be rule out since although Teavigo is caffeine-free, Polyphenon E contains 0.7% caffeine. Oral administration of 2000 mg/kg and intragastric administration (ig) Teavigo to rats resulted in lethality in 80% of animals treated http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00019. Histological analysis revealed hemorrhagic lesions in the stomach and intestine. Intraperitoneal administration of EGCG to mice resulted in dose-dependent lethality beginning at 150 mg/kg http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00016. Lethality was associated with increases in serum ALT levels, suggesting the involvement of hepatotoxicity.
These data suggest that high doses of EGCG can induce toxicity in the liver, kidneys, and intestine. Toxicity, especially in the liver and kidney, appears to be correlated with the bioavailability of EGCG. In the rat, where bioavailability is low [absolute bioavailability (F) = 1.6%], toxicity is confined to the gastrointestinal tract following po http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00020. In the dog, where bioavailability is much higher, hepatotoxicity, nephrotoxicity, and intestinal toxicity were observed. Toxicity was greater in fasted, than in prefed, dogs http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00019. The AUCplasma (area under the curve) in the prefed dogs was 19.8 g h/mL as compared to 205 g h/mL in fasted dogs following administration of 300 mg/kg, po. Recent studies in humans have also demonstrated that fasting increases the bioavailability of EGCG http://pubs.acs.org/cgi-bin/sample.cgi/crtoec/2007/20/i04/html/tx7000515.html#tx7000515b00021. Although there have been no reports of toxicity in human volunteers enrolled in intervention studies, careful monitoring of liver and kidney function is required until the risk of toxic events associated with tea catechins is established in humans.
[/quote]
Maybe this should be in the Superfood thread, but lets try it here.
With the above in mind, I just realized that Superfood includes green tea (extract?).
I have used Superfood quite liberally in my shakes so far, and was wondering on how much GTE actually is in this product, or what amount is considered safe.
Anyone from Biotest (or someone who otherwise know) care to shed some light on this?
-C
Johns Hopkins’ Se-Jin Lee recently found that overproduction of one myostatin inhibitor pumps the mice up even more than previous results instead of 60% up to 81 percent in females and a whopping 116 percent in males. Results of human clinical trials are pending.
Any updates?
[quote]GetSwole wrote:
I’m sorry the green tea calculations confused me, how many grams of EGCG would be needed per day?
8g of GTE at 50% meaning 4g of EGCG, or 8g of EGCG per day?[/quote]
4 grams of pure egcg or 8 grams of 50 percent extract. That’s to be taken 4 times a week to match the study using injections.
How long (total) have you been doing this again?
I’m interested to see how things are going.
Give us some updates man! lol
I’ve been taking about 2g/day of egcg…but wanna hear about this stuff!
Come a long way. It’s all about the cocoa
Anyone know if EGCG taxes the liver at all?
[quote]B rocK wrote:
How long (total) have you been doing this again?
I’m interested to see how things are going.
Give us some updates man! lol
I’ve been taking about 2g/day of egcg…but wanna hear about this stuff![/quote]
How long did you take it? Was there any noticeable effects?
hopefully this EGCG stuff works I just ordered a bottle of it.15 bucks with S/H.