Summer Blast and Coming Off 2 Years of Tren

Cialis works for 24-36 hours

Onset of action isn’t quite as rapid though. You still need to be aroused on it to get hard, though the level of stimulation required to trigger erection is minimal relative to baseline.

Priapism can lead to tissue death in the peen if not corrected

Welp, idk if its just time, or placebo from dick pills or the fact that i take 1/4 anastrozole a day now(not E2 related, i think, my body just likes anastrozole) but my libido is muuuch better… Not only erection, but the whole idea and me being interested. Its not as high as it was on huge blasts but it is pretty high for this poverty cruise im doing now. Very happy with my overall feeling, libido etc now.

When i get my hands on slin(i have to wait for prescription and it takes some time, which is a major bummer now) if i will feel any benefit of it, i will drop from 0,7ml of test E3D to 0,5ml of test E3D which would total me a 290mg a week.

Im also doing tons of cardio and i have lost around 14lbs for now. My cardio has improved, but strenght is exactly as shit as i expected it to be, so its all good now.

3 Likes

Pinned my first actual cruise dose today of 0,5ml(125mg) of test which ill be doing E3D for idk how long. Will do some bloodwork sometime later, to see how everything is going.
My cruise sums up to 290mgs of test a week, which is not low, but i have found lots of info that says that cruise dose should be 1mg per 1lb of weight and rounded up from there, so for me its 250-260lbs and by rounding it up i get 0,5ml E3D for conveniece sake.

What im doing for training now is - i have EOD strenght and EOD cardio days, so around strenght training i am doing insulin pre and post workout, and on cardio days i dont do slin, but i do 4iu of GH in the morning and then i do my cardio and also i fast till like 5PM.
So my bro-logic here is that i optimize my strenght training with slin and the next day i maybe lose some fat slowly while working on improving different aspects of cardio. I know building muscle one day and burning fat the other is an internet myth, but i am taking insulin and GH which could make this work at least for a little while. Anyway, will see how it goes. At least im on low AAS. At least as low as i am willing to go.

Which bloodmarkers are the best to see if i am somewhat ok on cruise?
Is it possible to have good HDL while on such cruise dose?
And what other markers should i wait to see to know that i am in somewhat of a good shape to start thinking about a new blast?

1 Like

Yes… Genetics…

You’re on statins, boosts your chance.

My HDL doesn’t seem to budge at all on higher test. I haven’t taken more than 125mg t/wk in over a year. But I have bloods… 100mg/wk, 200mg/wk, 250mg/wk… Doesn’t make a lick of difference.

It’s always borderline low. Between 42-45 mg/dl

On synthetic androgens however it’ll drop 20-70% dependant on the compound/dose. Any orals = lipid destruction…

Statins seem to hold my bad cholesterol in norms. The good one is shit tho. Best i had recently was a bit below 1mmol/L where it should be over 1mmol/L.

Edit - at my worst my HDL was 0,57mmol/L… the refference says it should be 1 or over. I had my best a few weeks ago at 0,87 i believe.

I’m no expert but…

It’s not just “the number” it’s also about ratios, particle size, trigs, insulin sensitivity, oxidative stress etc.

You could have say… HDL 0.7, LDL 1.8, total cholesterol 2.5 (on say high dose rosuvastatin), awesome particle sizes, low trigs or whatever.

A HDL of 1.2, LDL of 6 (random example) is almost certainly going to be worse

at worst with tren and tons of other shit my LDL was 3 or a bit below when my HDL was 0,57…
My best in most recent bloods was HDL 0,80 and LDL 2.07…
Im just interested in those not to be where i am under a super high risk of dropping dead :smiley:

Depending on what dose of statin you’re on statins can lower LDL by upwards of 50% and increase HDL by around 10%

So without the statins lipids would have a almost certainly been far worse.

Like… Your lipids are marginally worse than mine. I take a miniscule dose of statins… Just because (like 10-15 mg simvastatin cuz without it my lipids aren’t trash but they’re borderline)

My HDL is between 1.1-1.2 depending on when you test, what I’ve been eating at the time, exercise protocal etc. LDL is 2.3-2.4, total cholesterol is generally below 4, trigs below 1.

Off the statins my LDL is between 3.4-3.9, HDL 1.1, trigs between 0.8-1.4, total cholesterol between 5.4-5.7

Hence why I prefer using a tiiinnny dose of statins. Granted I don’t really watch what I eat… But it’s easier to just so this as opposed to adhering to a strict diet. My father lowered his total cholesterol from like 6.3 to something like 5.3 by going vegetarian… Fuck that

My mom lowered hers from 7.5 to 5 via low dose statins, weight loss, going vegan and a few other modifications. I used to think perhaps both had familial hypercholesterolemia, but one managed to acquire sub acceptable lipid panels through diet alone (still eats a ton of junk food though) and the other through like diet, some weight loss (was and still is obese) and like… 10mg of a high intensity statin/day.

LDL dropped down below 2… On 10mg rosuvastatin? It’s almost certainly not FH, just an absolutely atrocious lifestyle.

Even RYR powder for me on bloods works just as well as prescription grade statins! I recall one of my red yeast rice products was recalled in Aus, I was sent a letter warning me it had lovastatin, could cause rhabado etc… No shit… Why do you think people take RYR

concerns of citrinin contamination was what convinced me to switch from RYR to statins. Unfortunately personal use of statins is illegal here… In the country I’m planning to go to however meds like statins are legal to import for personal use

My original dose after hospital was 80mg of atorvastatin, but i have reduced it myself many years ago already down to 20… i have lifetime prescription for 80, so i just cut every tab in 4 pieces.
I was under the impression that they tax liver, so i tought its best to have it as low as possible.
So if i decide to blast, maybe i could up them to 40mg?

Wow… They don’t even recommend that dose here due to the risk of myopathy!

I’Ll bUy iT oFf YoU iF We EvEr MeEt (jokes). I take simvastatin, though I’d be keen to try rosuvastatin.

Does taking statins fuck up your liver?

And is 20mg a day a high dose? And should i up it to 40mg when i decide to up the test and stuff?

Transient elevations in LFT’s are sometimes noted. Serious hepatotoxicity is very rare unless combined with other drugs. Like prolonged use of oral AAS with statins is ill advised.

The antioxidant, anti-atherogenic effects re statin use are quite potent. There are mechanisms by which statins prevent plaque build up that are entirely independent of LDL-C reduction.

Testosterone itself according to the literature available isn’t particularly harsh on lipids relative to synthetic androgens. You’re looking at say, a 10-40% decrease in HDL-c depending on dose. I know you like to use high dosages, taking like a gram of test will probably drop HDL by around 40%. Below 600mgs and the median drop in HDL is only 20-30%

Combine test + ai and the drop in HDL is exacerbated, estrogen modulates glucose and lipid metabolism. Shifting androgen/estrogen ratios tends to fuck up cholesterol more.

Look at the data re lipids and c-17aa AAS. They’re all very harsh on lipids, but oxandrolone and stanozolol on a mg/mg basis is quite a bit harsher than methyl testosterone is re HDL depression, LDL elevation. Perhaps stanozolol/oxandrolone stimulates hepatic lipase activity moreso than methyltestoerone… But the androgen/estrogen ratio theory also factors in.

DHT derivitives tend to be the worst for cholesterol… That and tren, tren is the worst injectable, followed by virtually all orals AAS aside from maybe methyltestoerone (going by literature)

Interesting fun fact. In Japan AAS are legal, but needles are strictly regulated. Some health store products claiming to promote increased libido actually have a decent dose of methyltestoerone in them, can be seen on the label.

Look up kinjyasei, it’s a sexual health supplement sold OTC there, like herbal boner pills and whatnot.

This claims to cure ED and impotency… The catch? It contains 3mg methyltestoerone per pill, instructions are to take like 3-4 pills/day

Does it work? Probably… But not because it has a special formula if herbs…

But don’t worry… The product states it’s not appropriate for use if you’re under the age of fifteen :rofl:

I really wish this was more well known. It’s like one of the worst combinations you can run and guys who love orals tend to have bad cholesterol, so some percentage of them will use a statin to treat it. No bueno.

Shocker!! Guys who love orals also generally take a bigger hit to kidneys long term, and there’s the risk of hepatocellular carcinoma, peliosis hepatis etc. It’s not likely by any means, but if you’re one of those guys who likes to damn near permablast on orals I mean… Literature pertaining to prolonged use of methyltestoerone, fluoxymesterone etc was indicative hepatocellular adenomas and whatnot weren’t exactly uncommon. Plenty of the time said tumours were benign, but if you’re using a ton of orals for a very long time you could run into issues re hepatotoxicity, carcinogenicity and whatnot.

C-17aa AAS are actually still FDA approved for TRT, however they’ve predominantly been phased out due to concerns pertaining to the hepatotoxicity, carcinogenicity, dyslipidemia, impaired glycaemic control etc.

Do I think they don’t work? Probably… I reckon methyltestoerone probably does resolve symptoms in androgen deficient men, it’s even marketed in Japan (detailed in my post above) as an aphrodisiac for older men. Supplement states it is unsuitable for use in those below the age of fifteen though, so wE hAvE tHaT sAfEgUaRd. But the safety profile is inferior relative to testosterone.

You wouldn’t be able to find a correlation with dyslipidemia and guys who use orals BEFORE they take the orals. But c-17aa AAS within an acute context fuck lipids

There are a few alternatives to stootins (that’s how I say statins now)… Can probs take Bile acid sequestrants on orals… Can reduce LDL by around 30%. LDL of 190mg/DL is still better than 270 :joy: (thinking of the really harsh orals like superdrol)

Pcsk-9 inhibitors won’t be commercially available for a very long time… If ever…

I’ve taken statins and orals at the same time. You’re talking about a very short duration of use, like 4-7 days on three separate occasions. LFT’s were slightly elevated, but nothing insane.

Stootilydootilydootins… Got a bit of cabin fever going on… Week… God knows of hard lockdown… Thank god I’m getting out next year, I presume I’ll never come back at this point. Just need to tough it out until I have enough :money_with_wings: moneyz to leave. Australian citizens aren’t allowed to leave the country, but if I’m leaving the country to go live somewhere else that point is mute.

1 Like

Where you headed?

Not going to disclose openly. Shoot me an email if interested

1 Like

Just curious. I’ve always kinda thought I’d move to Aus one day; interesting when someone from there leaves. I know you’ve talked about why you made that decision before

5 kilometre radiuses, going on day 220 of stay at home disorders, drone surveillance, shooting protestors with rubber bullets, arresting and imprisoning them… Over the past year a cascade of mass surveillance bills have been footed through parliament

Premier’s have been shutting down parliament because it is too dangerous to congregate when there’s 30 cases per day

Etc …

Three years ago I would have set “Australia has it’s drawbacks, but if you like knowing you’re within a safe paradigm… If you want an amazing albeit very expensive standard of living so long as you don’t break any laws (possession of steroids in QLD/NT= potentially a twenty year stint in prison) Aus is a fantastic country”

Now? We are a bio police state… Shut out and isolated from the rest of the world… And domestic travel indefinately.

3 Likes