Hey guys, I’d like to request the shedding of light upon something I’ve never fully grasped regarding carb-restricted diets because in the words of Chris Shugart, I’m an idiot.
So, as I understand from the AD, your insulin sensitivity INCREASES because intake of CHO is less frequent, and it’s only logical.
However, I’ve read from several places (I think Question of Strength with Poliquin mentioned this) that restricted carb diets DECREASE insulin sensitivity, but I’m not exactly sure why. The logical deduction in my mind is that CHO is no longer a prominent part of the diet and insulin doesn’t get its ‘exercise’? Suffice to say, this particular notion doesn’t make complete sense to me.
In addition to where ever else I read it, I just read something in JB’s Getting Unshredded article:
[quote]4) Exaggerated Insulin Response
Lower carbohydrate diets can lead to short-term insulin insensitivity in the face of higher carbohydrate intakes. Now, that’s not such a big deal while still on the lower carb diet. After all, insulin sensitivity doesn’t have to be so good when carbs are restricted. But, when you start eating carbs again, this exaggerated insulin response can be a problem.
Oh, it’s not so bad for the first day or two of higher carbohydrate intake. The depleted liver and muscle glycogen stores will greedily gobble up those carbs. But remember, although the carbs are being gobbled up by the muscles and liver, there’s still an exaggerated insulin response. And with high insulin comes a reduction in fat mobilization and oxidation.
So even while the muscles and liver are filling up, there’s an anti-lipolitic effect. And once those glycogen stores fill up, if the carbs aren’t introduced slowly and the insulin controlled, fat gain can be rapid.[/quote]
He says insulin sensitivity is temporarily decreased? Yet, when returning to normal CHO intake, the insulin sensitivity is way elevated. Does this have anything to do with the empty liver/muscle glycogen stores? What I’m deducing is that insulin sensitivity is LOWER because the glucose goes STRAIGHT to its open sites to replenish glycogen without ‘hanging around’ in the blood too long. Now, the insulin sensitivity is HIGH (which makes logical sense in the first place) when normal CHO intake is present because the glycogen stores are filled and blood glucose functions as it “normally” would.
Could any of you provide a critique of my thought process, corrections, elaborations, etc.? I’d just like to get this physiological aspect sorted out.
EDIT: By the way, one more quick question, does sodium have ANYTHING to do with actual adipose tissue (formation, reduction) or is it merely related to water in smoothing out your physique?