The fact you fucked up you’re HPTA axis isn’t particularly due to age. AAS induced hypogonadism is a possibility with anyone who uses, not to say young people should be using, as neurologically the effects can be rather profound (though not as severe as say… the cognitive stunting induced by alcohol or the reward pathway remodelling induced by nicotine, cocaine etc) and many young people aren’t aware/can’t grasp the long term health ailments potentially induced by these compounds when used at high dosages and thus decide to use very high doses with little regard to overall longevity.
A singular testosterone injection (supra dose, long ester) is typically enough to suppress LH/FSH almost entirely (can link data to this). The ideology one can make gains as fast as an older dude on cycle simply isn’t true. A healthy teenager in his prime may have a TT of 800-1300ng/dl, perhaps even more (I recall one dude on here worried about having accidentally taken sarms… he got his T tested and it came out at like 1500ng/dl (don’t remember free T, but it was pretty high). A grown man on cycle will typically have a TT of 3-5000ng/dl+ with free T 5-10x + the top end of “normal”, that being said adults don’t have the same GH/IGF-1 output when compared to teens of which greatly contributes to overall anabolism and capacity to gain muscular mass… BUUUUT AAS themselves greatly increase IGF-1.
I find the cardiovascular pathology the most concerning, there has been a myriad of deaths recently, mostly young bodybuilding competitors in their 20’s with death causes as “unknown”, one appears to be an overdose on pills (suicide attempt), I’d hypothesise the other is cardiac related. It isn’t just anabolics however, the ones that die right after a competition, electrolyte imbalance, use of diuretics, improper use of insulin, clenbuterol, stimulants, beta 2 agonists, DNP must all be taken into account. However I firmly believe anabolics at high enough doses (and literature somewhat backs this up, though to what extent is disputed) will lead to cardiovascular dysfunction down the line and exaggerate the (cardiac) hypertrophic stimuli induced from exercise, of which by itself when taken to excess appears to be a high risk factor for atrial fibrillation… which itself can lead to clotting (strokes, myocardial infarction)
Then again, cigarettes, alcohol, stimulants, eating badly etc all have the potential to induce long term cardiac dysfunction, many get away with it… some don’t, it’s Russian roulette with genetics. There are ways to greatly reduce risk factors, but then again genetic predispositions many a times can’t be overcome, if you have congenital dilated cardiomyopathy/severe familial hypercholesterolemia you bet you’re ass you’re going to run into complications without medical intervention (statins, ace inhibitors, beta blockers etc)
Both my grandmas smoked frequently (one about a pack a day for 30+ years, the other a few a day for a couple decades), they are respectively 95 and 86, both my grandfathers died young due to tobacco related complications (myocardial infarction and emphysema) at 49 and the other in his sixties. Genetics are a cruel bitch, my genetic analysis (when uploading said dna, I got one of those tests) appears to indicate I have genes strongly indicative for longevity… which is a bitch as I can’t imagine my chronic pain will get better, I can only see downhill from here, being Ronnie Coleman status at age 60 will be an absolute and utter irritant. Should clarify the one that smoked a pack a day for a while is the one that’s now 95… My great uncle made it to 102! (or was it 101)
Interestingly my grandfather was over 6 foot tall (one was 5’6 the other 6’1)…