Sounds good to me, I have over 25,000 posts on the subject over the past 10 years. Some of our docs have been doing Urology and TRT for 25 years. I have debated the subject with doctors all over the world.
I look forward to sharing my knowledge with others and learning from them at the same time.
@increasemyt - I donāt think Iāve suffered from anemia, never been tested.
Honesty, this is the first time Iāve had my own blood work done outside the doctors officeā¦ And itās waaay higher than what I see from the doctor actually. These are all I can find online from my doc right now, from Jan 2019:
!Screenshot_20190701-121013|690x175
My HTC has been 45 every time Iāve tested it. The only other time I had estradiol checked was last December (non sensitive) and it was 25.6
I was moving up from 120mg a week to 140mg during this timeā¦ I think maybe around 130mg a week.
But would my T/E numbers jump that much from January upon moving up in dose to now being stable at that new dose? Numbers seem high going from 120mg a week to 140mg a week.
Also, my SHBG dropped from 38.6 to 27 from January to now?
Donāt even know which numbers to use as the baseline, haha.
Just saying your HCT is really low in comparison to your T levels, so something is out of wack.
Have you ever had HA1c tested?
When men are on testosterone their HCT is usually the lowest at 48. So for you to be at 44 with that high of a free T is strange.
So I wonder if there is something else outside of the TRT going on, cause your TRT numbers donāt look terrible.
I will say in older men, really high free T like that gives them issues a lot of times. How old are you?
@increasemyt - Sorry, Iām 42.
Iāve never had HA1c tested. Donāt even know what that is and will Google it now.
Iām still perplexed why my total T almost double between January and now going up 20mg a week.
Is it possible the most recent lab work from my outside blood work is incorrect? I wonder if they still have a sample they could retest.
I have an appointment with my doctor this week. Iāll get blood there to compare and ask about HA1c too.
Thanks again for the time, I appreciate it.
Yea tell him you want the full panel, full thyroid, reverse, antibodies.
HA1c, CRP, PGR, PSA, DHEA-S
I am sure I am forgetting a few but those seem the most important to me, feel free to add to the list.
Edit: Yea T levels continue to climb a lot of people donāt realize this, that is why it is usually a constant battle of adjustment when starting TRT for the first year.
Thank you. My PSA has always been good. Was 0.53 in December.
Iāll be sure to ask about others, much appreciated.
HA1c will tell you about how your insulin is functioning. The CRP is for inflammation in general.
My guess would be your E2 is tad high and that is being affected, chances are that and the extremely hi T are whatās doing that. Testosterone and estrogen play a large role in the management of glucose.
Your symptoms match those of someone who has insulin issues, or a pituitary tumor, or both.
This paper in no way relates to using a AI in men on testosterone. I would like to see your studies supporting a healthy level of estradiol in men who are actually on testosterone. I mean this is the population of men we are looking at correct?
So you would like to see studies of men on testosterone and aromatase inhibitors showing what?
That is irrelevant to the discussion, the discussion is specifically whether an AI or low E2 is responsible cholesterol management. I have proven, without a doubt, that E2 is in fact responsible for this role.
So are you saying taking anastrozole will always wreck your cholesterol regardless of what your E2 is?
This is not a hard concept to understand, as shown by the literature, not sure why so many are having trouble with it.
Would an increase of 10% testosterone cause a 10% increase in e2?
E2 causes breast cancer? Prostate cancer? Hmmmmm I wonder then why in all the studies in women where we give estradiol it decreases their risk of breast cancer as well as the recurrence rate in women who have been treated for breast cancer.
I wonder why we can treat men with prostate cancer with Estradiol. Shouldnāt that make the cancer grow like crazy? When we chemically castrate men for prostate cancer but then at the same time give Estradiol it dramatically improves their lives.
facepalm
Women take aromatase inhibitors for breast cancer to totally erase estrogen in the body. They take 1-10mg per day, this is why the literature shows altered bone resorption and cholesterol, because their E went to 0.
They give men estrogen for prostate cancer because it is super suppressive, it has nothing to do with estrogen combatting the cancer.
In fact I am glad you brought that up, because treating men with prostate cancer by chemical castration is terrible. You are making the cancer worse or progress.
New literature, in the last decade, proves that not only does proper testosterone levels reduce your risk of prostate cancer by 50%, it doesnāt matter if it was endogenous or through exogenous treatment.
So yea for 70 years we cut mens balls off, and it was the exact opposite of what we wanted to do.
This is all because of a single study in 1938 by Charles Huggins that deemed testosterone aggravated prostate cancer. Which makes absolutely zero sense because then every 21 year on the planet would have prostate cancer.
What happened is they aromatized and that actually caused cancer progression but they didnāt think estrogen was important to a male so they didnāt even look at it.
Any more questions?
Specifically, the current study found that 38,570 of the men whose records were examined developed prostate cancer between 2009 and 2012. Of these men, 284 had prescriptions for testosterone replacement therapy before they were diagnosed with prostate cancer. Their records were compared with 192,838 men who did not develop prostate cancer, of whom 1,378 had used testosterone therapy.
Researchers noted that while their initial analysis showed an uptick of 35 percent in prostate cancer in men shortly after starting therapy, the increase was only in prostate cancers that were at low risk of spreading and was likely a result from more doctor visits and biopsies performed early on. The authors stressed that the long-term reduction in aggressive disease was observed only in men after more than a year of testosterone use, and the risk of prostate cancer did not differ between gels and other types of preparations.
Testosterone Therapy Does Not Raise Risk of Aggressive Prostate Cancer, Study Suggests
you havenāt and cant provide a single study supporting āmanaging estrogenā in men that is beneficial when he is on testosterone. Working at a T Mill doesnāt make you a expert as your post prove you are not. Whenever a website doesnt list who their treating physicians are it always a T MILL
You can claim whatever you want, my posts speak for themselves.
Yours do too.
Your posts have not demonstrated anything. yeti308 is also correct in stating that none of the physicians are listed. We deal with many T Mills and this is the first sign of one in every case we have dealt with. If the physicians are well known and reputable, why do they need to remain secretive? Why is it that the docs we deal with clearly indicate who they are and what their qualifications are?
yeti308 has made very good points that you have not addressed. Why is it when we give E2 it treats prostate cancer yet you state that it causes it?
I already explained that, it would help if you actually read my posts. Estrogen is given is because it is far more suppressive than testosterone, so the goal is to chemically castrate the patient.
Besides, I have already explained how chemical castration increases your risk of aggressive disease, so your post is pointless.
My post where I ask why all the physicians are secretive is pointless as well?
Thats just how we do it. Every single one of our patients sees one of our doctors virtually before any prescription is written. So they all know our doctors.
Our medical director Dr Matt actually did a question and answer about 3 years ago on our blog that you are more than welcome to go read.
But no we do not just hand out our physician information, then everyone would go straight to the physician for everything and overwhelm them. We manage the patients for the physician, and any one of our clients can request a VHC and get seen within 24 hours by their doctor. Our physicians donāt do just this, they work in the ER, have multiple Uro practices in NY, we even have RNās.
Why I have to explain this to some newb is beyond me but there it isā¦
Are you implying yeti308 is a noob? You clearly have no clue who youāre speaking to. The few hundred thank you messages I have in my inbox from guys Iāve helped, that I do completely on the side of my regular business (for free) also demonstrates that Iām hardly what is considered a noob at this either. I have the privilidge of getting to deal with the best in the business.
You have done nothing but attack me personally with ridiculous fallacies and have not cited a single piece of literature backing up a single one of your claims.
That plus your very sub standard understanding of estrogens role in the metabolism of glucose and cholesterol tell me yes, you guys have a lot to learn.
The comment was directed at you BTW.
