Decanoic Acid Instead of Deca for Joints

We’ve all heard the praises of deca and it’s beneficial effect on joint inflammation. I’ve always wondered if it’s the nandrolone that helps with inflammation, or is it the capric(decanoic) acid ester that has the anti-inflammatory effect. It wouldn’t seem to me that if one were to inject nandrolone suspension that it would have the same benefit. On the other hand, it stands to reason that sterile capric acid probably would have some benefit, just like fish and flax seed oil have a beneficial effect on inflammation.

Capric acid comes from coconut oil, and I already know people who take coconut oil orally for inflammation. I would like to experiment with this, since I have bad shoulders, lower back etc. Anyone have any experience or know a good source for pure, sterile capric acid injection?

From what I’ve read so far, Deca has a masking effect at best and is bound to make your ligaments thicker, but decreasing collagen cross-linking integrity resulting in more brittle and weaker ligaments.

Regarding inflammation: chronic inflammation warrants using anti-inflammatories, acute inflammation is the first step in wound healing, though. Taking anti-inflammatories during that phase compromises wound healing.
This is a big misconception both in injury treatment, both in and outside of the sports realm.

If you’re interested in using agents to speed up healing, you might want to look into Oxandrolone, GHRP-6 and HGH.

Cool question, one I’ve often wondered as well. I remember reading a study where they injected a tendon with nandrolone decanoate and exercised it through various ranges of motion using various stresses. I’ll see if I can dig it up somewhere. The result was a smaller stronger tendon, seeing as collagen shrinks as it matures, makes sense.

What FattyFat said is interesting, if the mechanism that reduces pain is simply from the anti-inflammatory effect provided by the decanoate ester, then I’m wondering in what way it would decrease collagen cross linking integrity? Not a challenge to the statement, I’m literally curious.

Anyway if the ester is the culprit, shouldn’t testosterone decanoate provide the same effect? Also people using oral prohormones like M4OHN which is a derivitive of nandrolone, report joint pain reducing effects as well. So maybe nandrolone has a role to play?

Cool stuff anyway. Also to the OP what FattyFat said about acute injury is basically it. The old RICE theory is the most retarded thing ever conceived. Acute inflammation is the body trying to heal itself, inflammation is still involved in chronic injuries, but not to the extent that anything can actually regenerate anymore, hence why it’s mostly only functional movement that causes pain once injuries become chronic, at rest you’re usually fine or not so bad, becuase of its orthopedic nature. Anyway, there are various growth factors, macrophagues, etc, involved in the process, I might type some of that out later.

You might want to look into prolotherapy, or regenerative injection therapy as well (same thing). It works by inducing microdamage into an injured ligament/tendon/joint capsule, etc, drawing growth factors to the area and initiating repair. Pretty effective in my experience. Got an ass load of studies on different subjects using various proliferants and growth factors to repair various grades of tears, and cartilage damage. Saved me anyway. Most orthopedic issues are poorly understood, in fact most general practitoners have to google their anatomy nowadays.

Anyway, good luck with your research, keep us posted if you find anything usefull.

[quote]vexeN wrote:
Cool question, one I’ve often wondered as well. I remember reading a study where they injected a tendon with nandrolone decanoate and exercised it through various ranges of motion using various stresses. I’ll see if I can dig it up somewhere. The result was a smaller stronger tendon, seeing as collagen shrinks as it matures, makes sense.
[/quote]
If they injected nandrolone decanoate into the tendon, it would be some kind of prolotherapy, thereby making it hard to determine how much the injectant might have contributed to the result of a smaller stronger tendon - unless they had a control group that received dry needling.

[quote]vexeN wrote:
What FattyFat said is interesting, if the mechanism that reduces pain is simply from the anti-inflammatory effect provided by the decanoate ester, then I’m wondering in what way it would decrease collagen cross linking integrity? Not a challenge to the statement, I’m literally curious.
[/quote]
I wasn’t precise enough in my above post regarding nandrolone. The studies I’ve read about nandrolone making ligaments and tendons thicker, but weaker (reduced collagen cross-linking integrity) referred to systemic use, not to direct application into incompetent connective tissue.

[quote]vexeN wrote:
Anyway if the ester is the culprit, shouldn’t testosterone decanoate provide the same effect? Also people using oral prohormones like M4OHN which is a derivitive of nandrolone, report joint pain reducing effects as well. So maybe nandrolone has a role to play?
[/quote]
Sorry, can’t really comment on this one.
There is a study where testosterone (don’t know what ester) was used as proliferant during prolotherapy, with promising results.

[quote]vexeN wrote:
Cool stuff anyway. Also to the OP what FattyFat said about acute injury is basically it. The old RICE theory is the most retarded thing ever conceived. Acute inflammation is the body trying to heal itself, inflammation is still involved in chronic injuries, but not to the extent that anything can actually regenerate anymore, hence why it’s mostly only functional movement that causes pain once injuries become chronic, at rest you’re usually fine or not so bad, becuase of its orthopedic nature. Anyway, there are various growth factors, macrophagues, etc, involved in the process, I might type some of that out later.

You might want to look into prolotherapy, or regenerative injection therapy as well (same thing). It works by inducing microdamage into an injured ligament/tendon/joint capsule, etc, drawing growth factors to the area and initiating repair. Pretty effective in my experience. Got an ass load of studies on different subjects using various proliferants and growth factors to repair various grades of tears, and cartilage damage. Saved me anyway. Most orthopedic issues are poorly understood, in fact most general practitoners have to google their anatomy nowadays.
[/quote]

I’ve had good results with prolotherapy.
Still, healing doesn’t happen overnight with this treatment modality and it’s important to strike just the right balance between nutrition, training and recuperation to make this work.
Prolotherapied connective tissue needs to receive just the right amount of pull, applied from suitable angles, to help with collagen formation.