[quote]bushidobadboy wrote:
So, given that he has excellent hydration in his other discs, the dessication is not due to age.
What is it due to? Please enlighten me; I’m all ears.
BBB
EDIT: To cut you some slack, so that you can save a little face, I can see where you might have got the idea that the OP actually has ‘dessicated’ discs. You read that “A loss of fluid in the discs, as seen on MRI, indicates that the discs have become dry with age”. Or something like that.
But what you failed to grasp (a little knowledge being a dangerous thing and all that) is that in this case, there is perfect hydration in all the visible IVDs, except for two. This is a clear marker that his spine is not dessicated (since dessication is age-related and refers to a near-uniform loss of discal hydration) but instead that he has lost the nuclear fluid from 2 IVDs.
Since the healthy IVD is a sealed unit, the nucleus cannot just disapear for a holiday, to return later. No, the fluid is either there (intact, healthy nucleus), on its way out (a bulge), left the disc (extrusion) or has left, but shut the door behind it (sequestration).
Yes, dessication may be referred to as degeneration and a discal lesion is also degeneration, but the two are very different beasts, since dessication will happen to everyone, irrespective of lesions in the disc, eventually.
I await your reply, oh student of the spine. Better bring your A game though.[/quote]
i dont need you to save me anything boy.
you’re a complete moron. how dare you spew conjecture and nonsense to this guy who is scared as shit about having a surgical or otherwise permanent back condition?
how dare you tell him he’s guaranteed to have a protrusion yet lack the full set of axials to make this call? did he PM you the DICOM file?
what is this “nuclear fluid” you speak of? Do you have any idea what disk desiccation is? that is what he has. And guess what, it happens at 4/5 and 5/1 more so than other levels.
You seem to be unable to grasp that a lack of fluid signal within an intervetrebal disk (on both T1 and T2 weighted images) means there is less water there. Sorry, its a condition related to senescence. It does not mean the nucleus pulposis has “left the building”. its simple pathophysiology.
You need to review your extrusion/sequestration definitions, too. See, those are big words that make the audience clap for you. I dont care if i get the applause. I just dont want to fuck the OP. The annulus fibrosis has torn in an extrusion and the NP is no longer restrained. When it breaks free of the parent disk its a sequestration.
And you’re very sorely lacking on your understanding of degenerative disk disease. Your “sealed unit” of a disk isnt actually sealed. Because you’ve lost signal and/or height, that does not mean a giant hunk of NP has herniated. Loss of disk height happens. Period. Your sealed unit does not herniate into the endplates, it does not herniate into the canal, it does not herniate. Again, pathophysiology. And again, you’re very wrong in saying “dessication may be referred to as degeneration and a discal lesion is also degeneration, but the two are very different beasts”. Actually desiccation is part of the spectrum of degenerative disk changes. one in the same. Differing levels of severity. Much like the progression of annular tear to protrustion to extrusion etc is a spectrum. You would have been correct (for the first time) in saying that degenerative disk disease and disk “herniations” are different beasts. Yet you maintain they are linked, and loss of disk height indeed implies the latter.
if this is your A game…well.