The Predator Program

a small number of anecdotes are not sufficient data and my first guess would be she suffered from metabolic syndrome.


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G-Flux! It’s been 14 years or so since I last saw Berardi bring the topic up. Does he actually still support that theory?

There was a professor at Cornell doing a study on a diet. When he presented his data, he explained the results didn’t match expectations because the participants lied about what they ate. In other words, the study can only prove himself correct.

The participants lost about 25% of the weight they were supposed to. Coincidentally, meta-data analysis on diet studies show that diet studies regularly produce results of weight loss that are about 25% of expectations. Now either people all magically lie about how much they eat in the same exact way, or the way we do our calculation is wrong.

Going back to a study mentioned earlier, the NIH and Dr. Kevin Smith determined weight loss is nonlinear. So is it a coincidence that we see deviation in expectations? Or is Dr. Kevin Smith correct?

I’m not really sure what this has to do with anything we’ve been discussing, but my guess would be a) the vast majority do not stick to the diet as prescribed and/or b) the vast majority do not accurately measure their caloric intake. I’ve never met a layman, so to speak, that pulls a scale-out and weighs their food while on a diet. Most people don’t have a clue what a tablespoon or cup or pound actually looks like and underestimates their portions, in my experience.

Weight loss is most likely non-linear for numerous factors. Weight loss does not always meet expectations for numerous reasons.

If weight-loss is nonlinear it means that weight-loss is not dependent on caloric consumption. By all means I’m in agreement that it does influence weight, it’s just there’s other factors that can change weight in both directions.

My point bringing that up, was to support that changes in weight are still not accurately understood. It is therefore possible changes in weight were not due to metabolic syndrome.

The flaws in tightly coupling caloric surplus or deficit to weight change is that the body is not a simple machine. The body can choose to store or get rid of that for a variety of reasons. This is also supported through gene conditioning by leptin receptors as demonstrated through Dr. Ruby Leibel’s research. If gene conditioning on leptin receptors can influence weight, what other gene conditioning factors can influence weight?

No, it doesn’t.

*It just means there are other factors at play as well. Fat gain/loss much like muscle gain/loss does not occur in a vacuum.

Fair enough, that by itself does not mean weight-loss is not dependent on calories; however, literature on nonlinear weight loss do not always credit the calorie as a dependent factor. For instance, the set point theory…

Gonna be honest, I think the set point theory is really dumb.

*Should clarify, I don’t think the theory or the idea that the body maintains a range/set point is dumb. I mean, if I don’t eat as much as I normally do I get hungry… I think the idea that human beings are bound to said range/point is dumb.

Yeah, that’s generally true. “Nutrient dense” is essentially shorthand for “high nutrient density per calorie”, but there are some calorie-dense, nutrient-dense foods that could trip people up. Mostly fat sources like avocado, nuts, whole eggs, etc.

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Oh, sure, absolutely.

I think there is a lot of evidence towards epigenetic influence and gene conditioning on weight/health.

Take for example studies on birth and adult weight after World War II. Areas that were famished and women gave birth, the children had both low birthweight as a child and subsequently reduced weight as an adult. The reduced rate was disproportionate to caloric intake as an adult. This suggests that the body may be conditioned genetically to reduce weight in order to reduce caloric maintenance. Similar studies on vegans has resulted in similar findings.

No this brings up a very interesting point… The converse maybe true. Women who eat excessive amounts of food and give birth to a high weight baby, the baby is more likely to be obese as an adult. The odds of a 10 pound baby being obese as an adult is somewhere along the lines of 70%, but I’d have to look up the stat again.

Of course, findings can be dismissed by respective diet and habits they get from their family.

I do believe in epigenetic influence on weight due to the gestational diet. That being said, I also believe the epigenetic influence can be undone throughout life.

Edit: I forgot to add, I do believe this is part of the contributing factor towards the obesity epidemic too.

In my n=1 experiment, I took my deadlift from 405 pounds to 600 pounds in about eighteen months. I didn’t do anything like eat all bananas for a month though, or eat raw meat every other day. I just went to the gym, did some deadlifts, went home, ate some meat and vegetables, and repeated that a bunch of times.

PureNsanity, riddle me this: have any of your n=1 experiments achieved comparable results in strength gain, fat loss, or literally anything at all indicative of progress towards a meaningful goal?

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Just so I’m clear here fellas, is it safe to gather from all of the collective knowledge being dropped, that generally eating mostly nutritious food may have a generally positive affect on health and weight? I’m not a scientist or anything, so it’s possible I misunderstood.

:green_apple:+ :corn: + :poultry_leg: = :grinning::boy::dancer::muscle:

:hamburger: + :fries: + :cake: + :doughnut: = :nauseated_face::older_man:t2::hospital::ambulance:

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Is learning how to control vital bloodwork not meaningful?

I honestly cannot believe that you consider yourself sufficiently equipped to give people advice on anything diet and fitness related.

I feel bad for the people you’re conning into thinking that you’re some sort of authority on any of these matters.

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So when someone with 120 fasting glucose comes to me and asks, “how can I reduce my fasting glucose?” And the advice that I give them drops their A-1 C and fasting glucose levels to normal ranges, you think they’re being conned?

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So I was just sitting around the other day and I found myself wondering how I could possibly reduce my fasting glucose levels…

S

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Why in the fuck are people approaching you for this advice in the first place unless you’ve advertised yourself to be an expert? Not once in my life has anyone approached me asking how to drop their A-1 C, but I don’t go around giving anyone the idea that I’m fully equipped to help them with that.

THAT’S the con. I’d think someone with an IQ of 140 should be able to see that quite clearly.

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People that I work with, family, friends, etc. ask me on occasion. I’m not promoting it… Regardless, the point is they achieve the goal they want and I’m not charging them a damn thing.

No I don’t see how that’s a con.

I just explained how that’s a con, but you’ve glossed right over that just as you’ve glossed right over every other valid criticism leveled against you. Since you’re awful dense for a 140 IQ, I’ll spell it out for you again…

WHY??? Why are these people asking you these very specific questions?

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