Hello friends,
Before i post i would like to preface it with my now customary flame liability clause.
“I am NOT trying to re-invent the wheel here, this is a question not to see if an already fantastic protocol/system/product/idea can be improved upon, who am i to assume such a thing? Merely a question to see if it can be adapted to suit my own personal needs more effectively - thinking outside the box as it were…”
Now the legal stuff is out of the way…![]()
I get severely surpressed by the 19-Nor tests… Nandrolone i have had experience of, but i am assuming Tren too.
I love Nandrolone, and aggression aside, i would love to use tren too. (I am even bad on Deca though FFS!)
I have reasoned that it isnt estrogen related, nor androgen related, but i think i have a particular sensitivity to HPTA suppression via progesterone/Progestins, possibly through its interaction with prolactin.
a) In which case i would favor a 6-8 week stasis, would you agree?
Would an 8+ week stasis provide any more benefit/recovery than a 6 week stasis? Also would it be wise to run a SERM through the 100mg Stasis period-in order to maximize the therapeutic potential of the stasis?
b) Also when tapering, the normal protocol is “80,60,40,20” with a week on each dose.
Would it be more favourable for a tough recovery to do something like:
Wk1-80mg
Wk2-70mg
Wk3-60mg
Wk4-50mg
Wk5-40mg
Wk6-30mg
Wk7-20mg
Wk8-10mg
And maybe run a low dose (20mgTam) SERM throughout until the end or even 1 week past the taper, with another week at 10mg?
OR maybe run the taper with 2 weeks at 80mg, 2 weeks at 60mg, etc… so the body has a chance to normalise to the new dose over a 2 week period rather than 1 week - which i suspect could be too fast when in a very suppressed situation…?
And the third question, Would Cabergoline be indicated for such a suppression?
Would it be an ideal perk or PCT addition/replacement? Seeing as progesterone does have an effect on prolactin* and excess prolactin is thought to cause impotence and loss of libido through reduction of FSH and Gonadotrophin releasing hormone. (i believe its action in men is yet to be understood - but it has quite a few considering all the effects from modulating it with a drug such as Caber…)
Thoughts gentlemen?
JJ
- From what i have found and can understand so far progesterone ANTAGONIZES the estrogen induced release of prolactin from the pituitary. ONLY when the progesterone is administered within 1hr of the estradiol however, when it was administered 4 hours after it had no effect on the prolactin release.
From what i understand about our use of progestin based(?) AAS, progesterone acts to agonise the effects of estrogen… could this possibly cause a higher amount of estrogen induced release of prolactin, which in turn is NOT antagonoized by the progesterone as the necessary dosing protocol isn’t in place?
This is just a thought, there are so many mechanism as work here it is extremely difficult to pinpoint anything at all - especially for me who left education at 15…!
JJ