Androgenic drugs have a mechanism in and of themselves that is suppressive, as shown in (Winters, et al. 1979). The study also showed in the doses they used, that concurent use of clomephene citrate was able to nullify this suppression, as well in addition to of course testosterone, which aromatises to E causing suppression.
It is just simply my argument that based on the evidence, that you can use steroids of low androgenic rating along with clomephene citrate, midcycle as a way to increase FSH/LH secretion and untimately increase testicular size via metabolic increases to the testes. This of course is an alternative to using HCG, and you all know my opinion of that.
As for the other arguments let me remind all once again that the studies I listed did infact show no hpta suppression while using 100mg/week of test E, in conjunction with clomid use!!!
now I do and always have included the use of an antiE in the protocol to keep estrogen levels in the normal physiological range, to minimize rebound post cycle, and I have talked about tapering it down as you reach the 50mg/ week mark so that you are completely off it at the 25mg/ week mark of test E, which research shows completely no suppression of the FSH/LH.
Now I have no clue where Anthony gets his logic from but I bet it is due to the fact that he as absolutely no formal training in physiology or pharmacology.
If recovery of the hpta is all about regaining natural homeostasis, then how does adding a whole wack of unnatural drugs achieve this? The truth is it doesnât do it any sooner or at all.
You have to clear out all metabolites of non testosterone drugs from your body before you can even hope to obtain HOMEOSTASIS (anthony, you should look up this word in the physiology text that you never read)
That is what the waiting period is for. It doesnât matter what pct drugs you use if there is still artificial metabolites in your body you cannot even begin to recover your natural homeostasis - no matter what Anthony says, this is just a fact, there is no getting around this.
His method would be to use hcg during this time period which I say would make you feel better, but just royally fuck up your hpta even more.
My method allows you to feel good during this peroid, but instead of screwing things up, actually helps because as the weeks go by your body is able to adapt back to a lower amount of testosterone in the body.
Once you begin the taper, provided There are no other hormones left in your body besides testosterone that could suppress your hpta, there is proven to be nothing holding the body back from producing FSH/LH at 100mg of test E per week while using 100mg and concurently as long as E is kept in check and obviously the lower you go the less suppresion, so you all get my point I am sure.
The argument is the fact AR seems to thing that steroid users are hypogonadic. I dissagree. The bottom line is if you were eugonadic before suppression with endogenous AAS then you will be eugonadic following, provided you donât fuck up your system by adding a whole wack of other pct drugs such as hcg, lupron, e.t.c.
If woman can be on birth control for years, which, essentially does the same thing as what steroids do to men, and then have kids, following, then why are men considered 'hypogonadicâwhile on steroids?
Hypogonadism is a disease process where the testes is damaged and is incapable of producing normal amounts of sperm and testosterone.
Now if this is what occurred during suppression, then the effects would be permament, as disease causes scaring and fibrosis, and no drug would reverse this.
In normal Males who have have low testosterone/ sperm production, not attributable to any form of AAS use, when HCG is administered and normal amounts of sperm and testosterone is produced by the testes, then the testes is deemed to be eugonadic, and the problem is most likely elsewhere such as the pituitary or the hypothalmus.
So if you can respond to hcg, there is no reason why you canât respond to your own LH secretion, and therefore there is no need for hcg use, as it will be more suppressive and harmfull to a eugonadic male then good.
Now since studies have shown that testosterone administration in low doses to have absolutely no harmful side effects, and when given in sub physiological levels so that total T is within physiological normal range, the body as a whole will view itself as OFF CYCLE. At this point since there is evidence that shows no harm to the body, it does not matter how long you taper for. You are technically off AAS. So the Argument of quiting cold turkey so full recovery can occur sooner, is irrelevant. By using the argumentâs method, you quit cold turkey, then rely on drugs to kick-start the natural testosterone production back into gear. Meanwhile there is a test crash, and libido is lost. (hormone crashes are unhealthy by the way - so score a point for tapering here)Now using the HCG recovery protocols: the user will have been in a âcrashedâ state for quite some time{ as he will have to have waited untill all drugs had cleared before he could even have begun recovery), and then will have to have waited even further until the hpta was somehow restored. Using HCG during this time period as a bandaid approach may relieve the symptoms of a test crash, but would ultimately cause lower testosterone levels -unaided by any drugs for quit some time to come.
By using the waiting period, along with the test taper, you avoid all these problems and smoothly come off the test without ever having to wory about your testosterone levels falling below natural physiological norms.
So if you want to talk about retention of gains and safety and efficacy -The user who tapers will always be either above his normal level or at his level of test production. He dosenât have to slow down in the gym. He doesnât have to pick up a bottle of viagra, and he wonât suffer from withdrawal symptoms that include mood disorders, dramatic weight loss, fat gain, excessive acne, not to mention that loss of libido, can be a real relationship killer.
whereas the user of hcg will need to inject quite frequently to match the taper approach, and adding more drugs does little to achieve natural homeostasis - you are just rather shifting dependancy from one drug to another.
Now AR can disagree all he likes, and nit-pick about facks here or there, however he canât alter human physiology to suit his argument. This method works, and more and more members are going to attest (pardon the pun:)) to this as time goes by.