Right because I specialize in other areas and have never studied medical until now.
My knowledge is sufficient to tell me why I don’t block estrogen until it’s absolutley necessary. I have numerous doctors that I could use as a reference, but then again you probably will continue arguing.
Go in acting like an immature nuisance and I’ll cotnue spreading helpful info and try to stay away from endless arguments.
In days and years to come I will have a big smile on my face when you realize how wrong you’ve been.
No one said to inhibit (you don’t even understand the difference between a blocker and an inhibitor) estrogen if it wasn’t necessary, thats what I have been trying to explain to you, so you won’t be confused anymore.
You and your cohorts claim that aromatase inhibitors alone are responsible high cholesterol, decreased BMD and suboptimal fat loss. This is blatantly false, and I have explained the differences multiple different ways.
It is about the level of e, not anastrozole.
Studies on older men taking 1mg a day (way too much) of anastrozole with no testosterone barely show a decrease in BMD.
You are telling me someone with optimal E and Vitamin D levels are going to have bone resorption? That makes 0 sense.
Currently there is no literature to recommend the use of AIs in adult males to treat late-onset hypogonadism, obesity-related hypogonadotropic hypogonadism, gynecomastia, or male subfertility, although some positive effects have been reported. The adverse effects on bone health seen in females treated with AIs are not seen in males.
Height gain [mean (SE)] at 24 months was: AI, +14.0 (0.8) cm; GH, +17.1 (0.9) cm; AI/GH, +18.9 (0.8) cm (P < .0006, analysis of covariance). Height SDS was: AI, -1.73 (0.12); GH, -1.43 (0.14); AI/GH, -1.25 (0.12) (P < .0012). Those treated through 36 months grew more. Regardless of treatment duration, height SDS at near-final height [n = 71; age, 17.4 (0.2) years; bone age, 15.3 (0.1) years; height achieved, ∼97.6%] was: AI, -1.4 (0.1); GH, -1.4 (0.2); AI/GH, -1.0 (0.1) (P = .06). Absolute height change was: AI, +18.2 (1.6) cm; GH, +20.6 (1.5) cm; AI/GH, +22.5 (1.4) cm (P = .01) (expected height gain at -2.0 height SDS, +13.0 cm). AI/GH had higher fat free mass accrual. Measures of bone health, safety labs, and adverse events were similar in all groups. Letrozole caused higher T and lower estradiol than anastrozole.
CONCLUSIONS:
Combination therapy with AI/GH increases height potential in pubertal boys with ISS more than GH and AI alone treated for 24-36 months with a strong safety profile.
I’ve increased my T to 0.4ml/3 times a week with no adverse reaction to my prostate, the PSO and the Passionflower are doing their jobs right now. Not sure what type of epiphany my wife went through but we been having a go at each 2-3 times a day for the past 2 weeks and I’ve been able to achieve my goals each time. Hurray
The downside to the increase in T is the 15 pounds I’ve gained in the past 2-3 weeks, not liking that much. Starting cutting back to 0.3 ml / 3 days a week of 200mg/ml see if that helps.
I know, I was just pointing out that you had given us enough info to know. I don’t think of it in terms of mg usually, just what mark on the needle to fill to.
I have heard of many people having great benefits, beyond erection strength, with 5mg of cialis daily. I have not tried it as my erections are already ‘cialis grade’ now that my testosterone levels have been optimized. For some, optimized testosterone levels still result in poor erection strength which could be a vascular issue or something else entirely. In these cases, cialis has helped. It also has benefits for improved blood flow and cardiovascular health all around.
I have stayed away from any types of supplements, other than a high quality protein powder, as supplements in general always have some strange effect on me.
