hi im 30yo 181cm tall and 85kg(lifting for 10year)
my 1st and 2nd cycle is test only with 1st 250/week and 2nd 300/week
i was thinking to do my next cycle with :
test e 300/week 1-12
dbol 20mg/day(oral) 1-4
stano 20mg/day 7-9
stano 40mg/day 10-12
adex .5 (every injectionday starting week 2)
PCT (2 week after last injection)
tamo 40/40/20/20
clomid 50/50/25/25
i was wondering if i should just drop the Dbol or not??
Naaah, the safest protocol is the safest protocol but the best is probably the worst one in terms of safety.
Anyway, as far as “safe” - there are 2 schools of tought. One would agree with you saying that you should better up the dose on 1 compound. But there is the other one, which believes that the higher the dose of 1 compound, the more sides of this compound, and they would prefer smaller doses of many compounds in hopes to not get the sides of any of them, instead of blasting 1 drug.
But 250 test is really too low for anything. If i was a beginner i would probably do a few months on 500, then a few on 750 and then think about adding stuff.
Should also differentiate someone trying AAS for the first time versus someone with some dose response info under their belt.
At least to start 250 mg/week protocol will help someone (albeit small minority) that may have unknown pre-existing heart condition, autonomic dysfunction, Ca+2 handling, etc learn important info with less risk. I am probably in that small minority but I am glad I patiently pyramided the dosing rather than jump in the deep end right off the bat. In hindsight, once you know your response then perhaps starting at 200-250 mg/week seems silly. But of course that is the luxury of hindsight. Once you have that data and you are ok you can move up.
What causes more harm? Five 500 mg/wk cycles, or one with Test, Tren and Anadrol? I don’t know the answer to that? Just something to think about.
I think larger blasts (I don’t cycle) can make sense. More of the caloric surplus goes to muscle, less to fat. Less to cut after, which results in keeping more of the gains (which were more than the lower blast).
I am not doing the second blast I proposed. I’ll probably just stick to cruising. I think I am basically to the point where I can’t hold blast gains on cruise (or not that much of them). If I do much else, it would be increasing cruise dose, or adding in 4 week oral blasts. Just doesn’t make sense being bigger for 4 months, to be back to about baseline in another 3-4 months.
I believe that we dont know what causes more harm…i have seen here grown men fucking themselves up by doing cycles i did when i was 17 and i didn’t even know what caloric surplus was or what pct is. I believe one has to look after his health and just go by actual data about it, because some can ruin themselves with 500mgs and some can be on grams forever, like Mark Felix and Stan Efferding.
I think we can only talk about what causes more harm to the specific person. Smth that causes harm to you in 1 year might not even raise my BP and the other way around as there are non smokers with lung cancer and then there is my fat mom with her 100kgs and smoking whole life and she has never had a health problem.
Without a doubt, the safest way to protect yourself against lung cancer is to avoid smoking cigarettes, and yet, at the same time, it’s also true that not all lifelong smokers are doomed to develop cancer.
In fact, the vast majority don’t. Scientists have long wondered why, and a new study adds weight to the idea that genetics has a role to play.
Although lung cancer risk among smokers is dependent on smoking dose, it remains unknown if this increased risk reflects an increased rate of somatic mutation accumulation in normal lung cells. Here, we applied single-cell whole-genome sequencing of proximal bronchial basal cells from 33 participants aged between 11 and 86 years with smoking histories varying from never-smoking to 116 pack-years. We found an increase in the frequency of single-nucleotide variants and small insertions and deletions with chronological age in never-smokers, with mutation frequencies significantly elevated among smokers. When plotted against smoking pack-years, mutations followed the linear increase in cancer risk until about 23 pack-years, after which no further increase in mutation frequency was observed, pointing toward individual selection for mutation avoidance. Known lung cancer-defined mutation signatures tracked with both age and smoking. No significant enrichment for somatic mutations in lung cancer driver genes was observed.
I dont know. I just stated two popular beliefs but we have no idea which one is more correct.
Anyways your test dose is so low you are not getting full benefits so i would up the test. I believe that if you dont take enough of a drug you dont get its full benefits. For me at lean 250lbs i believe the limit is like 700-1000mgs of one drug. I would add another compound when im closing in on that dosage. I would use orals only for the cosmetic benefits as i dont think any oral is anabolic enough to outweigh the potential liver damage and heartburn.
I have always thought stacking AAS is the best for adding muscle. I didn’t use extremely high dosages, but my cycles usually amounted to between 500 and 600mg/wk
For example:
200mg/wk testosterone cypionate
200mg/wk Deca
20mg/day Winstrol
Just another point of view.
(Please note that all my AAS were pharmaceutical grade.)