[quote]enchilnada wrote:
[quote]seekonk wrote:
[quote]Prisoner wrote:
the research showed no hpta suppression while using a serm and low dose testosterone - 100mg per week
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I am trying to understand what this means. Is it from a published study?
Did someone measure endogenous T production during treatment and determine that it was unaffected? This would require some way to distinguish between T from exogenous and endogenous source, maybe by some calculation. Or does it just mean that endogenous T returned to baseline immediately after cessation of treatment?
What dose of SERM are we talking about?
[/quote]
There are studies which are supportive of different aspects. For example, Winters et al., found that if subjects are administered 100 mg of clomiphene twice daily, LH levels are unable to be decreased (and in fact continue to rise) even when serum testosterone levels are elevated up to over 2,000 ng/dl (i.e., the equivalent of the peak concentration reached after a 200 mg dose of testosterone enanthate). Naftolin et al., also had similar results.
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interesting stuff…
if this is accurate, why don’t we use Clomid on cycle to prevent shutdown altogether?
presumably, this would be due to a limited time-frame, and it no longer works? i’m curious if that’s why the TRT folks don’t use this…
IMHO, these are interesting studies, but using them here is a tad flawed in logic, as we’re not talking about someone having a normal HPTA system, but one fully suppressed from a cycle.
EDIT: as far as Teslac, well, it was awesome… basically the first AI, as it had close to a 90% suppression of estrogen, while bumping testosterone up by 47%…