Low T, Low GH, Hypothyroid Possible, Metabolic Issues. Hopeless in Australia

Ahahaha, nah 250mg e3.5d.

So before i blasted i cut, for 3 days before i started cutting i was taking 80mg of fuseaminde, dropped all my water, then i spent the next 7 weeks cutting on 500cals a day and 125mg e3.5d. I lost 6kgs over those 7 weeks.

I then started my blast, im in my 7th week of 250mg e3.5d. At first i put on quite a bit of water, but i combat that with the water ill do 80mg of fuseaminde for 2 days every 2 weeks and only lose 1kg.

No sides, atm which is great. Im max only eating about 2200cals perday sometimes more some times less.

Im now thinking of ordering something else to stack for the rest of my blast. Either

Tren A
Anavar
Winni

Thanks for the feedback. I don’t really know anything about all this. But I do know my doc isn’t treating me correctly. He talked about loading up (250 every 3 weeks) for a while and then stretching out the shots once it’s built up. So he obviously has no idea. Had bloods done after 2.5 weeks and i was at 11nmol. Just hoping for a better doctor in Brisbane.

@trebor1

General protocol doctors prescribe is

125mg every week

Then we as a collective work together on tnation to help each other figure out the best protocol for each other.

Generally its something like 65mg e3.5d

Is the furosemide prescription or black market. Diuretics can really fuck with you
 BADLY if taken for bodybuilding purposes (in the absence of extreme oedema). Electrolyte imbalances can cause fatal arrythmias and dehydration resulting from diuretic use can cause Renal Failure (esp if using it while cutting).

This is you’re first blast, you’re already using 500mgs/wk and you want to add either trenbolone, oxandrolone or stanozolol. I’ll break down each compound for you, explain why I don’t think they’re great ideas however it’s you’re choice, in the end it all relates to whether you care about longevity or whether you’re “here for a good time, not a long time”.

Trenbolone is a derivitave of nandrolone (19-nortestosterone), on paper and in action it is exceedingly strong, being 5x stronger than testosterone mg/mg in both anabolic and androgenic potency. With this strength comes more risk, more side effects and
 more gains (but is death in fifteen years from now worth gains? There’s a case report or two of heart failure developing after as little as 1 year of tren usage). Side effects of trenbolone are numerous, and include

  • Cystic acne
  • Hair loss
  • Lactation (prolactin related)
  • Agression/moodiness (part from androgenicity, part from dopamine depletion, decreased serotonin from 19-nors)
  • Tren cough/ cardio intolerance (why does this occur? I have no idea, however tren appears to cause respiratory distress)
  • high blood pressure
  • destroyed lipid profiles
  • Kidney damage
  • anorgasmia (prolactin)
  • erectile dysfunction (prolactin)
  • insomnia
  • gyno (mechanism I don’t understand properly, prolactin and progestin receptors do exist in breast tissue though, and tren increases prolactin significantly + has progestogen if activity)
  • liver toxicity (tren displays some degree of hepatic resistance to metabolism)

While these side effects aren’t all guaranteed, it’s likely you’ll experience some of them. Seriously if you aren’t a competitive bodybuilder I wouldn’t recommend touching tren, but do whatever you feel is best to live the best life possible. Tren doesn’t aromatize btw, nor can it be 5a reduced into a dihydro metabolite.

Stanozolol (winstrol) is a C-17AA derivitave of dihydrotestosterone. It is notoriously harsh on the lipids, 6mg/day almost cuts HDL (good cholesterol) in half (decreases HDL-2 subfraction by up to 80% on that dose), and raises LDL by quite a bit. Much of this detrimental effect on HDL (I think) is due to increased hepatic lipase activity (catabolises HDL) as all C-17AA AAS tend to have a marked detrimental effect on the lipids but stanozolol seems to take the cake. Stanozolol tends to cause joint pain for unknown reasons, possible PR receptor antagonist, Progesterone has anti-inflammatory properties, stanozolol also appears to make tendons more prone to tearing when lifting heavy. The C-17 methyl group in stanozolol makes it liver toxic, case reports of liver failure have been noted in people using stanozolol, even at therapeutic doses (although it’s very unlikely). As an anabolic agent stanozolol’s actions are fairly independent of the AR, it has an amazing ability to increase nitrogen retention more so than most other AAS (which is a good thing up to a point
 Don’t want kidney damage), however aside from the temporary cosmetic benefit the risks are quite high.

Oxandrolone is another C-17AA derivitave of dihydrotestosterone, I’ve actually used this one before. Studies show ox is faked around 66% of the time in UGL preparations, so either get it tested or be very confident of the UGL you’re using. I’m fairly sure my oxandrolone was mixed with something else given the stimulant type feeling, very high blood pressure and extreme increase in HR it gave me at merely 25mg/day. Despite what many say, oxandrolone isn’t “mild” it’s anabolic activity is very strong, however you won’t gain any water weight on it, all weight gained will be pure muscle. Uniquely, despite being C-17AA oxandrolone isn’t as hepatotoxic as other C17-AA compounds as a large majority (also unique) is metabolised via the kidneys (probably makes it nephrotoxic tho). Oxandrolone isn’t safe however, it tends to be free of visible side effects, but it will DESTROY you’re lipid profile (actually it depends on the individual, but some guys get HDL in the single digits on 40mg+/day. Blood pressure may also become an issue.

Finally I assume you’re aware of the cardiac complications that can occur as a result of AAS use. Given you’re labs on 250mg/wk, you’re not blasting and cruising (no offence), but you’re blasting and blasting. Adding in even more is fairly likely to cause complications, esp if you use recreational drugs on top of the anabolic steroids and eat badly. If you’re okay with all this then go ahead, feel free to email me if you want any advice though (email in profile description). You can cut fine with just test, if you absolutely had to add something why not a non C-17AA DHT derivitave at a low dose. If you want a more detailed description of the above compounds just ask and I can give details about pharmokinetics, metabolites and whatnot

From my GP I get 50 per 3 months. And yes they do deplete electrolytes but its also potassium sparing so you need to monitor your potassium levels and intake.

V8 juice is great to replenish

Yes i would be using a low dose of caber to manage this. No leaky nips for me no thanks haha. My mrs still has leaky nips 2 years later after a kid. Haha

My happy trt dose is 250mg e3.5d. Thats why i went to 500mg.

This is a fair point, however my doctors are happy at my trt dose albeit quite high.

My good you have some knowledge, i love reading your posts, i have read this many times over but you can never read this information enough.

I want to come to a decision tonight, been thinking about it for the past few weeks. My ugl has a great sale on which ends tomorrow night i think.

I mean, my advice is to stick with test, but otherwise if using additional agents I’d stay away from orals and tren

Hi Mate - very interesting thyroid results. From my (admittedly limited) research, the jury is still out on the significance of rT3. I’ve had my thyroid tested by multiple different doctors, including an endo, and have never had rT3 panels. Each time I’ve been tested for TSH, FT3, FT4 and antibodies, but never rT3. The reason my thyroid has been so thoroughly examined is because I had thyroiditis about a year ago, which appears to have self-resolved (all of my blood work looks pretty good) but I remain symptomatic. Insomnia has been debilitating, but I also have anxiety, fatigue and a few other things. My doctors do not believe my thyroid is to blame for these symptoms, but I’m sceptical (particularly because another cause has not been found).

I might ask for rT3 to be tested in a few weeks when I next have my appointment. I am also thinking about getting a 24 saliva cortisol test done privately.

I also had low free T, so 7 weeks ago I started TRT and my labs look pretty good, although my progesterone is low (I’m not sure if this could account for my symptoms). While I feel improvement on TRT, I do not believe that all of my symptoms are referable to low T - I believe another cause is at play. Particularly because I’ve been experiencing low level non-location specific pain in and around my neck/sinuses. It is all very strange TBH


I would definetley get RT3 checked out! Can also have it done easily through Imedical if the doctors give you grief!
Unfortunately trying to get to the bottom of this stuff can be an absolute shit show!
I’m feeling better since going on T3 but still not 100% same as you


@theninja are you still seeing the doctor on Glenferrie Road? (I can’t remember the practice’s name, as it went through two or three in quick succession.)

How are you finding it?

I’m back in Melbourne soon, and was considering taking a look.

Hmmmm 50/50 big sell at the start but I’m unsure atm
 I questioned a few things and he didn’t take it to well
 still better than anywhere else I’ve been to


Thanks. Keep us posted on how things go.