Thank you sir, means a lot. I’m pretty hard on myself, as I’m sure most of us are. I still see myself as the 135lb 24 year old in my head. Still feel like I’m 20lbs too small.
You say this, but there are other threads where it was said deadlifts don’t build shit. Where were you in those threads? I’ve done deadlifts for years and didn’t notice any muscles getting bigger. Surely something was growing as I was able to up the weight over time.
Deadlifts lack an eccentric portion of the exercise (unless touch and go is implemented), the eccentric portion of an exercise is the portion of which most muscular micro trauma occurs. Furthermore, in terms of overall back development, theoretically the rack pull would be superior, as it allows one to pull more weight, the stretch the trapezius muscles get during such a lift (with heavier weight) should theoretically lead to more hypertrophy, thus in terms of back development, the rack pull is superior (I think).
Exactly, I feel as if RDL’s activate the glutes and hamstrings to a far higher degree, with less detriment to the CNS, thus allowing one to perform more volume. Squatting after deadlifting is a fairly taxing endeavour.
@unreal24278 interesting, I was just thinking today that I want to do some rack pulls. I probably won’t deadlift again as I’ve injured myself 3 times now doing them. I’m thinking rack pulls will be safer.
I agree, I actually do not do RDLs and Squats typically in the same day. I do legs twice a week, and typically do RDLs and Squats on separate days.
I like the idea of rack pulls, but for some reason have never implemented them. Might add them to my workout for 12 weeks and see what happens. I’ve watched guys do em, thought to myself I should try them, but always forget.
That’s not a healthy way to look at it. And yet I know what you mean. Nothing will ever be good enough. So you just keep going. For me it’s just stupid because I don’t need to look good for my partner and I don’t want any other external validation. But I’m still never going to be happy.
Your rear delts are well developed. What do you do to bring them out?
Nope. It only takes one good swimmer to get the job done, but Clomid/Nolva on cycle is not going to stimulate anything on the LH front, and HCG certainly won’t either. The HCG takes the place of LH, so it would stimulate natural production, but your LH is going to essentially stay at zero.
I’m not going to recommend exercises as I don’t have the experience. However rack pulls aren’t as complex a lift comparative to deadlifts, thus the chance of injury is lower… as long as you don’t load up as heavy as possible and lift retiredly heavy for ego…
No kidding, it’s good and bad right? It’s what keeps us eating well and working out hard, but I definitely have body dysmorphia pretty bad, and wish I could better enjoy the work I’ve put in. I’ve always thought 215/220 I’d be satisfied. But I’ll probably never be able to maintain that while off AAS. I plan to genuinely run one more cycle and call it quits, but who knows if I’ll hold true to that.
I’m like you though, happily married, no real need other than it makes me feel good.
As for rear delts, I honestly don’t do anything special. High cable rear delt flies or seated bent over DB rear flies. I actually stopped doing all direct rear delt stuff for a year or two as I didn’t feel like they needed much focus, and now feel like they’re lagging a bit so just started doing them again.
To be fair, I’ve got pretty decent back development and I’ve never deadlifted heavy. I used to try bring my numbers up but i just suffered too many injuries. The bulk of my back work is just different types of rows and weighted pullups.
SB
Everyone @Singhbuilder @iron_yuppie @studhammer @weightliftingwithoutlimits @aaronca @theinneroh @yeet @floop
What are you’re resting heart rates on cycle? I’m asking because AAS are known to interfere with catecholamine uptake (increasing sensitivity to, up regulation of beta adrenergic receptors etc). Thus heart rate at rest and heart rate response to exertion is impaired. I have (mild) autonomic instability, POTS (postural orthostatic tachycardia syndrome) cross-linked with fibromyalgia and BHJMS (both fo which I have), so I take beta blockers (on cycle I typically double dose), yet today/yesterday I forgot to take and the increase in HR in response to standing up (and exertion) was somewhat scary (not dangerous though)… Made me a little bit depressed to realise my condition hasn’t improved at all and that I’m somewhat dependent on this medication to keep my heart rate down… but it is what it is… I’m asking the question about heart rates in relation to this
The avg AAS user had a RHR of 80+, 90 wasn’t even out of the norm (box plot, so represented was 25-75% of cohort). If my resting heart rate was 90 BPM I’d be profoundly uncomfortable/anxious, it’s around 45-60 most of the time… anything above 70BPM bothers me… Furthermore, look at the impairment in relation to post exercise HR recovery, demonstrating sympathetic nervous system upregulation.
I hypothesise this is potentially the reason all my sources sell benzodiazepines (that or for insomnia induce by AAS use)… however that’s retarded, as for insomnia melatonin (of which is a prescription only medicine here) or Benadryl should do the trick, furthermore if benzodiazepines are sold utilisement in relation to lowering HR/reducing anxiety induced by excess sympathetic nervous system activity… they should just sell beta blockers dammit…
My RHR does t change on cycle. My Fitbit always reads RHR 55-58.
Variable ONNNEEEEEEEEE, collecting daaatttaaaa yeet
(mine changes fairly quickly, it’s a difference of about 5-10BPM, pretty dramatic difference)
Depends on the compound though, Dbol doesn’t appear to change my pulse rate at all, nor does drostanolone, testosterone specifically tends (for me) to induce the largest change within pulse rate.
Don’t know why mine doesn’t. My BP avgs about 10-15 higher on systolic but not diastolic on cycle. RHR is always fairly low.
My BP is always waaaayyyyyyyyyy too low (beta blockers don’t make a difference)… I’m talking like 90/40 at times, nothing has managed to make a notable budge aside from oxandrolone (of which I don’t think was oxandrolone/might have been cut with something, gave me side effects of which would be expected from stimulant medication)
Dbol managed to raise my BP to around 100/50, actually I wonder if the absurdly low blood pressure has anything to do with the dramatic increase in Pulse rate when transferring from sitting to standing (orthostatic hypotension), as POTS is typically not associated with a drop in BP, however if my BP was to drop even lower than say… 90/40, it’d make sense, I’ll analyse that variable next time I check my BP.
Mine varies also according to the compound. On tren its 70 to 80 but on var and testosterone it’s usually around 85 to 90.
SB
I’m referring to cruise doses that are at or below physiologic doses, where you might get some FSH-stimulated spermatogenesis with or maybe without drugs.
Don’t be depressed about your HR. Taking a day off from your B-Blocker will cause a rebound increase in HR. If you have an autonomic issue and not afib, you should be on a 2nd gen B-blocker like atenolol, which is known for having less reflex tachycardia especially when you stand up.[quote=“unreal24278, post:51, topic:261782”]
uncomfortable/anxious, it’s around 45-60 most of the time…
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Sounds like the one you are on is working though. This is very low.[quote=“blshaw, post:52, topic:261782, full:true”]
My RHR does t change on cycle. My Fitbit always reads RHR 55-58.
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This is also low, but you’re older than me and I take Adderall. My RHR measured by fitbit is in the 70s and sometimes hits 81 when I’m feeling sick or its the day before an exam.[quote=“unreal24278, post:55, topic:261782”]
My BP is always waaaayyyyyyyyyy too low (beta blockers don’t make a difference)
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They shouldn’t. They are one of the last line treatments in HTN. ACEi/ARB + thiazide + calcium channel blocker then even spironolactone come before BBs in the guidelines. We usually don’t even use BBs unless there’s a compelling reason, like afib.
This absolutely is what is happening. Orthostatic hypotension is one way to experience reflex tachycardia. And like I said unless the BB you are taking is atenolol then it can be making your situation worse. You might benefit from reducing your BP meds (or changing to atenolol) and getting into the 110s/80s.
How do you deal with it? Anything above 75 makes me very anxious (currently 72 directly after eating a very large meal) postprandial increase in HR
How would you perform cardio with a RHR of 80+? HR would skyrocket, with post-exercise HR recovery impaired, theoretically you’d be looking at 100+bpm for a little while…
I guess perhaps it doesn’t bother most and I’m just prone to anxiety/hyperaware of my body?
Nonsense, not low at all for someone who trains like I do.
Answer From Edward R. Laskowski, M.D.
A normal resting heart rate for adults ranges from 60 to 100 beats per minute.
Generally, a lower heart rate at rest implies more efficient heart function and better cardiovascular fitness. For example, a well-trained athlete might have a normal resting heart rate closer to 40 beats per minute.
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@unreal you know more than my doctor lad. I’m in awe at your knowledge.