Cardioselective BB (2nd gen) have less of a tendency to cause ED (note it’s still possible, but not as likely)
I assume you’re thinking of clonidine off label for anxiety treatment… for many (myself included) clonidine can be overwhelming sedating (switched to guanfacine… still very sedating but can tolerate a low dose)
Perhaps (with a doctors permission) try out a cardioselective BB (if physiological side effects of anxiety are MORE than merely a fast heart rate, such as tremors, increased respiratory rate etc) then you’d probably benefit more from a first gen BB that targets beta 1 and beta 2 adrenergic receptors 
Otherwise non pharmacological intervention such as meditation and yoga can help quite a bit, as can music, going for a walk etc.
I’d perhaps get an echo beforehand though, I know HR is individualist and genetic in nature, but without beta blockers my RHR will still routinely hit the low 40s/high 30s during the night, you didn’t drop below 65… 92 isn’t a problem per se (still theoretically within normal) however I’d get an echo just to make sure.
My halter (during a period of frequent panic attacks) showcased a mean hr of 79, max of 175 (from wrestling my father lol) and min of 41
RHR is currently as we speak 67, because I’m on test mast and dbol (excess sympathetic nervous system activation)
Personally I don’t use high dosages of Atenolol, 50mg/day or so. You can figure out what dose works best for you. I find my HR drops by about 6bpm per 50 mg pill (but stops the insane acute spike in HR I can going from sitting to standing #autonomicdysfunction)… don’t use this as a rule of thumb though, for all you know one pill might drop you’re HR 30bpm
I can give you this advice. If you decide to try without a script, start slow, sometimes even 25mg/day can work wonders pertaining to atenolol
The likely reason for you’re hr issues is the AAS. Anabolics stimulate/upregualte the sympathetic nervous system, upregular beta adrenergic receptors and induce cardiac autonomic dysfunction (and predispose one to lethal arrhythmia via numerous mechanisms) at a dose dependent manner. Some like you or I (given I have autonomic dysfunction absent of any AAS) are more sensitive to this effect than others… hell, some can’t even use TRT without heart palps initially, it’s EXTREMELY varied in response