[quote]lothario1132 wrote:
What you do not understand is how the egg or the child came to be, and this is your stumbling block. :)[/quote]
No Lothario1132, this is your stumbling block.
[quote]
Unless you realize and learn what genetic mutation is, you will never understand how an organism different from its parents can be born.:)[/quote]
I know exacly what genetic mutation is and will explain the FACTS below.
[quote]
BIG HINT:
Remember those Down’s Syndrome babies I told you about who have more DNA per cell than their parents? They are real. There really are people with Down’s Syndrome. And they really do have a genetic difference from normal people. And their parents aren’t like them.
OPEN THAT BRAIN!!! :)[/quote]
Open that Brain? Ok.
Increased amounts of DNA don?t mean increased function. Biologists have discovered a whole range of mechanisms that can cause radical changes in the amount of DNA possessed by an organism. Gene duplication, polyploidy, insertions, etc., do not help explain evolution, however. They represent an increase in amount of DNA, but not an increase in the amount of functional genetic information?these mechanisms create nothing new. Macroevolution needs new genes (for making feathers on reptiles, for example), yet people who believe what you do completely miss this simple distinction:
Moreover, molecular biology has discovered mechanisms for genetic change that go beyond point mutations, and these expand the ways in which new traits can appear. Functional modules within genes can be spliced together in novel ways. Whole genes can be accidentally duplicated in an organism?s DNA, and the duplicates are free to mutate into genes for new, complex features.
In plants, but not in animals (possibly with rare exceptions), the doubling of all the chromosomes may result in an individual which can no longer interbreed with the parent type?this is called polyploidy. Although this may technically be called a new species, because of the reproductive isolation, no new information has been produced, just repetitious doubling of existing information. If a malfunction in a printing press caused a book to be printed with every page doubled, it would not be more informative than the proper book. (Brave students of evolutionary professors might like to ask whether they would get extra marks for handing in two copies of the same assignment.)
Duplication of a single chromosome is normally harmful, as in Down?s syndrome. Insertions are a very efficient way of completely destroying the functionality of existing genes. Now is the time for you to “open that brain”, lothario1132. Biophysicist Dr. Lee Spetner in his book Not By Chance, analyzes examples of mutational changes that evolutionists have claimed to have been increases in information, and shows that they are actually examples of loss of specificity, which means they involved loss of information (which is to be expected from information theory).
The evolutionist?s “gene duplication idea” is that an existing gene may be doubled, and one copy does its normal work while the other copy is redundant and non-expressed. Therefore, it is free to mutate free of selection pressure (to get rid of it). However, such “neutral” mutations are powerless to produce new genuine information. Dawkins and others point out that natural selection is the only possible naturalistic explanation for the immense design in nature (not a good one, as Spetner and others have shown). Dawkins and others propose that random changes produce a new function, then this redundant gene becomes expressed somehow and is fine-tuned under the natural selective process.
This “idea” is just a lot of hand-waving. It relies on a chance copying event, genes somehow being switched off, randomly mutating to something approximating a new function, then being switched on again so natural selection can tune it.
Furthermore, mutations do not occur in just the duplicated gene; they occur throughout the genome. Consequently, all the deleterious mutations in the rest of the genome have to be eliminated by the death of the unfit. Selective mutations in the target duplicate gene are extremely rare ? it might represent only 1 part in 30,000 of the genome of an animal. The larger the genome, the bigger the problem, because the larger the genome, the lower the mutation rate that the creature can sustain without error catastrophe; as a result, it takes even longer for any mutation to occur, let alone a desirable one, in the duplicated gene. There just has not been enough time for such a naturalistic process to account for the amount of genetic information that we see in living things.
Dawkins and others have recognized that the ?information space? possible within just one gene is so huge that random changes without some guiding force could never come up with a new function. There could never be enough “experiments” (mutating generations of organisms) to find anything useful by such a process. Note that an average gene of 1,000 base pairs represents 41,000 possibilities?that is 10,602 (compare this with the number of atoms in the universe estimated at “only” 10,80).
If every atom in the universe represented an “experiment” every millisecond for the supposed 15 billion years of the universe, this could only try a maximum 10,100 of the possibilities for the gene. So such a “neutral” process cannot possibly find any sequence with specificity (usefulness), even allowing for the fact that more than just one sequence may be functional to some extent.
So Dawkins and company have the same problem as the advocates of neutral selection theory. Increasing knowledge of the molecular basis of biological functions has exploded the known “information space” so that mutations and natural selection?with or without gene duplication, or any other known natural process ? cannot account for the irreducibly complex nature of living systems.