throughout my research i haven’t found anything conclusive about this issue. im trying to find out if pct’s like, Clomid, Nolvadex and HCG will help slow or minimize the effects of hair lose.
i know it kinda depends on how vulnerable you are genetically to getting hair lose. but im speaking in a broad general sense.
my cycle:
week 1-8 450 mg deca
week 1-10 600 mg cypionate
Clomid, Nolva Etc is not going to do anything to protect you from hair loss. It has only to do with your genetics and how predetermined to hair loss you are. There are products to help with the hair loss but its not any of those things you brought up.
I like to run my bread and butter with a ratio of 5:3. Therefore if you want to run 450 deca I reccomend you up the Cyp to 750.
5AR inhibitors like propecia & avodart work with test (on cycle), preventing DHT conversion.
They don’t work with all AAS though, sometimes they have no effect (var & dbol come to mind), at other times they aren’t recommended. Where they do work, they can actually cut down on gains a bit, too, since DHT is obviously a decent compound in its own right. Still, I use it with T because I like my lettuce.
In other words, stay tuned for someone else to tell you how they work with deca 'cuz that I can’t tell you offhand.
I have been pulling back a little and the test and winnie cycles I’m sure have not been helping… But I started using Nioxin shampoo and conditioner about 6 months ago and have noticed that my hair has become a little thicker and I have not been loosing as much hair in the shower hardly any actually… I don’t know all the scientific stuff about Nioxin except that you have to use it every time you wash your hair and that it gets rid of the build up and excess oil. So it might be worth a try?
thanks,
so does anyone have any other recommendations on products to use with the cycle to help with hair loss?
[quote]whotookmyname wrote:
5AR inhibitors like propecia & avodart work with test (on cycle), preventing DHT conversion.
They don’t work with all AAS though, sometimes they have no effect (var & dbol come to mind), at other times they aren’t recommended. Where they do work, they can actually cut down on gains a bit, too, since DHT is obviously a decent compound in its own right. Still, I use it with T because I like my lettuce.
In other words, stay tuned for someone else to tell you how they work with deca 'cuz that I can’t tell you offhand.[/quote]
Where i’m from Lettuce means Vagina. Do you mean Vag?
This has been answered (I’m gonna call him a moron in a sec… is it still allowed?! ;D)
ngggghh…
The stress of holding it in is tooo much… must flam…ee…hhhhhhhh…
pah!
Do you have present hair loss issues? You may have nothing to worry about.
I, personally, have zero hair loss issues on cycle. A lot of guys don’t. I can understand your wanting to be careful, but don’t spend too much money all at once.
By using a 5AR inhibitor with Nandrolone the following happens:
Nandrolones androgenic component is DHN. This is why Nando is such a sought after anabolic for its ability to illicit gains and is very very mild on the androgenic issues… All that good stuff we have come to know and love with test - aggression, acne, hair loss, prostate hypertrophy, bodily hair growth and enlarged clits (you know who your are…).
If we inhibit the 5AR enzyme then the nandrolone is not converted to this very mild androgen - which does attach to the scalp and other areas… but has a very very weak effect.
This allows more potent androgens to get to the site instead.
The bit i am confused with and i really would like BR to confirm this for me - where does the DHT them come from in the example given above? Is it from the test injected? If so wouldnt the 5AR inhibitor reduce the DHT too?
Or is it a case of it (the inhibitor) being used up dealing with the mild nandrolone conversionvleaving less of the drug to inhibit the testosterone conversion?
There is a good 50% of the answer i believe!
Brook
[quote] Brook wrote:
Where i’m from Lettuce means Vagina. Do you mean Vag?
;)[/quote]
lol just what sort of lettuce… no, what sort of vag do they have out your way But you’re right though, it can definitely imply that. Here in SoCal we occasionally use it in reference to hair haha.
[quote] Brook wrote:
…
The bit i am confused with and i really would like BR to confirm this for me - where does the DHT them come from in the example given above? Is it from the test injected? If so wouldnt the 5AR inhibitor reduce the DHT too?
Or is it a case of it (the inhibitor) being used up dealing with the mild nandrolone conversionvleaving less of the drug to inhibit the testosterone conversion?
[/quote]
I’d certainly like to hear from BR as well, but yes, unless I’m totally mistaken, you’d be getting some T → DHT conversion.
I’m the same as Cortes. I may have had gray hair at 18. But even on over a gram of gear I have no issues with hair loss.
[quote]whotookmyname wrote:
Here in SoCal we occasionally use it in reference to hair haha.
[/quote]
I’m in socal too…always nice to know of fellow tnation members nearby…especially ones who dabble on the dark side 
lol
/hijack
DG
[quote]TrainerinDC wrote:
I’m the same as Cortes. I may have had gray hair at 18. But even on over a gram of gear I have no issues with hair loss.[/quote]
I’ve honestly never even heard of it being an issue. I really don’t think it’s even something to worry about unless you are running a ridiculously high dose. No one I know has ever had any trouble and some of them are using 1.5g’s a week. I
[quote]Dirty Gerdy wrote:
whotookmyname wrote:
Here in SoCal we occasionally use it in reference to hair haha.
I’m in socal too…always nice to know of fellow tnation members nearby…especially ones who dabble on the dark side lol
/hijack
DG[/quote]
“dabble on the dark side”… wow, you sound exactly like an old friend of mine. Pretty sure I’m in the same county, too, based on a gym you mentioned a couple of days back (not sure why but I thought that you were in SD until I read that).
[/quote]
[quote]waylanderxx wrote:
I’ve honestly never even heard of it being an issue. I really don’t think it’s even something to worry about unless you are running a ridiculously high dose. No one I know has ever had any trouble and some of them are using 1.5g’s a week.[/quote]
Interesting. There sure seems to be a consensus amongst you guys on that, so maybe I just coincidentally hit a major hairline battlefront at the same time I was on an easy, T-only cycle. I am older though, so it wasn’t totally unexpected 
C’est la vie.
lol, thanks all. i dont mind the sarcastic responses.
well as you can see from my picture i have a full head of hair which is kinda more reason why i was worried in the first place because i know how nice it is and i dont want to lose it. i guess i agree with the consensus that i probably dont have to worry. personally i dont know anyone who suffered this issue but it was always a concerned me a little.
with that said, may the juicing begin
ps. i will show before and after pictures of my scalp ;o0
[quote] Brook wrote:
By using a 5AR inhibitor with Nandrolone the following happens:
Nandrolones androgenic component is DHN.[/quote]
Rather DHN is a metabolite of nandrolone, but it is not more androgenic. Less so actually.
Yes
The issue really is just that the amount of nandrolone is reduced. I don’t think the DHN acts as an inhibitor in any way. If it binds to the AR it will be effective: where it is weak is not after binding, but rather having less tendency to bind.
The AR is either activated or not: it’s not a question of some substances weakly activating it but rather that they leave more AR’s unbound.
If the example above is when using nandrolone, then DHT can come only from whatever natural or injected testosterone is present.
If the example is when using testosterone or just under natural conditions and using a 5AR, DHT comes from what portion of 5AR enzymes remain unbound by inhibitor but are binding testosterone at any given moment. Particularly in the case of finasteride a fair amount of this will always occur because it inhibits only one of two types of 5AR.
Binding to 5AR doesn’t use up inhibitor. It comes on and off, unchanged, and is inhibiting any given enzyme molecule when that molecule is binding inhibitor. It is used up only by metabolic elimination (I suppose principally or entirely by the liver followed by the kidneys but haven’t looked into it.)