I’ve been trying out yohimbine and caffeine the last couple of weeks with good results and a order of piracetam just got here, which I have not used yet.
So I was just wondering if there was any reason not to use yohimbine with piracetam…? Should I wait till I’m done cutting to use it?
Last weeks protocol:
Morning with Breakfast -
.5g DMAE
1.5g Piraceteam
10mg Vinpo
Throughout the day -
10mg Vinpo x2
1.5g Pira x1
I love how I feel on this set up. Very postive and awake and my reaction time is up quite a lot as well.
This weekend I got pretty blizted for the first time since before Xmas last year and had NO hang over at all. I mean, I didn’t feel ‘on top of the world’ but besides a bit of hunger and groggyness I was in great shape.
Gonna stop the drinking but continue the protocol.
[quote]Acodd242 wrote:
I’ve been trying out yohimbine and caffeine the last couple of weeks with good results and a order of piracetam just got here, which I have not used yet.
So I was just wondering if there was any reason not to use yohimbine with piracetam…? Should I wait till I’m done cutting to use it?[/quote]
Should be fine.
Blood sugar and memory
http://www.jonnybowden.com/2009/03/senior-moment-maybe-its-your-blood.html
I’ve been daily alternating Provigil (Moda) and Yohimbe-Free Spike for a couple of months now. I took Provigil daily for a week or so before this and YES it does lose its effect (for me). Spike did the same. But now that I alternate them by day, I no longer get sleepy in the afternoon and am able to teach Calc with a clear head. The Provigil is taken with breakfast and lunch, the Spike on an empty stomach before breakfast and before lunch.
Vinpo stopped working for me. I know cycling it is not a good protocol, so I stopped altogether.
This thread should be our first ‘sticky’ in here. Who’s with me?
Just ordered some Pyroglutamate and ashgawandha figured I’d see what your guys opinion on Pyro.
Does anyone know a decently priced supplier of aniracetam or oxi that accepts paypal, I’ve ordered 3 times from various places and they keep getting canceled.
me
[quote]bushidobadboy wrote:
Headhunter wrote:
Vinpo stopped working for me. I know cycling it is not a good protocol, so I stopped altogether.
You are positive about that? I can’t see a reason why that should be. Obviously ones enzyme profile may change over time accelerating clearance of certain compounds, but not to that extent I think. What dose did you work up to before you quit?
BBB[/quote]
I found the same thing. It had a huge effect on me when i started and its been around 6 months now taken everyday at 30-40 mg. I’m still going to take it though.
[quote]bushidobadboy wrote:
Headhunter wrote:
Vinpo stopped working for me. I know cycling it is not a good protocol, so I stopped altogether.
You are positive about that? I can’t see a reason why that should be. Obviously ones enzyme profile may change over time accelerating clearance of certain compounds, but not to that extent I think. What dose did you work up to before you quit?
BBB[/quote]
I started out with 20 mg/ed for 2 weeks and then 30. Maybe it was that my new norm was therefore not unusual so I didn’t notice anything. Been thinking of starting again.
Could Polycythemia Vera (type I) have anything to do with it? (too much blood production)
[quote]bushidobadboy wrote:
Headhunter wrote:
bushidobadboy wrote:
Headhunter wrote:
Vinpo stopped working for me. I know cycling it is not a good protocol, so I stopped altogether.
You are positive about that? I can’t see a reason why that should be. Obviously ones enzyme profile may change over time accelerating clearance of certain compounds, but not to that extent I think. What dose did you work up to before you quit?
BBB
I started out with 20 mg/ed for 2 weeks and then 30. Maybe it was that my new norm was therefore not unusual so I didn’t notice anything. Been thinking of starting again.
Could Polycythemia Vera (type I) have anything to do with it? (too much blood production)
Interesting question on the polycythemia. If it were secondary polycythemia, i.e. increased number of RBCs from non-pathological origin (e.g. TRT) then the increased O2 transport capability of the blood, might mimic some of the effects of vinpocetine, but not the enhanced ATP synthesis in the brain.
Are you sure you have type I (which is a pathological condition) and not type II which arises from non-pathological causes (high altitude, smoking, AAS, etc).
BBB[/quote]
It just hit me that I hijacked the thread. Can you PM me with more info?
I too think this should really be a sticky. It’s a fantastic wealth of knowledge.
Right about the end of 2008 I got pretty drunk with my cousin. I’d say a 10/10 as far as how drunk I’ve ever been. (I spent the night puking for the 2nd time ever). I woke up at noon still wasted, hung over all day, sick as shit all night and STILL feeling horrible the next day.
Fast forward to last weekend (3mo later) to a point where I had been using DMAE for 1.5mo and using Paracetem & Vinpo for 1.5wks and that’s where I was last weekend. I went out to the Cask with some friends and got pretty torn up (first time since the end of '08); probably 8/10.
So you would think that if a 10 landed me in the puke bin and all the wonderful after-effects that happened you would think that an 8 wouldn’t put me in the best place the next day(s). Wrong. Aside from feeling a bit tired for a few hours after waking; I was 100% fine. And I blame it on my Brain Function Booster. 100%
Both nights were full of drinking hard liquor and eating and then late night eating again. I weighed about the same as well…
Just thought I’d share my thoughts on some things that BFBs are doing for me. Not that I’m going to drink much more…but interesting nonetheless.
I think the OP would need to be updated with information if you stickied it.
Interesting study on Modofinil in JAMA recently:
Effects of modafinil on dopamine and dopamine transporters in the male human brain: clinical implications.
Volkow ND, Fowler JS, Logan J, Alexoff D, Zhu W, Telang F, Wang GJ, Jayne M, Hooker JM, Wong C, Hubbard B, Carter P, Warner D, King P, Shea C, Xu Y, Muench L, Apelskog-Torres K.
National Institute on Alcohol Abuse and Alcoholism, Bethesda, Maryland, USA. nvolkow@nida.nih.gov
CONTEXT: Modafinil, a wake-promoting drug used to treat narcolepsy, is increasingly being used as a cognitive enhancer. Although initially launched as distinct from stimulants that increase extracellular dopamine by targeting dopamine transporters, recent preclinical studies suggest otherwise.
OBJECTIVE: To measure the acute effects of modafinil at doses used therapeutically (200 mg and 400 mg given orally) on extracellular dopamine and on dopamine transporters in the male human brain. DESIGN, SETTING, AND PARTICIPANTS: Positron emission tomography with [(11)C]raclopride (D(2)/D(3) radioligand sensitive to changes in endogenous dopamine) and [(11)C]cocaine (dopamine transporter radioligand) was used to measure the effects of modafinil on extracellular dopamine and on dopamine transporters in 10 healthy male participants.
The study took place over an 8-month period (2007-2008) at Brookhaven National Laboratory. MAIN OUTCOME MEASURES: Primary outcomes were changes in dopamine D(2)/D(3) receptor and dopamine transporter availability (measured by changes in binding potential) after modafinil when compared with after placebo.
RESULTS: Modafinil decreased mean (SD) [(11)C]raclopride binding potential in caudate (6.1% [6.5%]; 95% confidence interval [CI], 1.5% to 10.8%; P = .02), putamen (6.7% [4.9%]; 95% CI, 3.2% to 10.3%; P = .002), and nucleus accumbens (19.4% [20%]; 95% CI, 5% to 35%; P = .02), reflecting increases in extracellular dopamine.
Modafinil also decreased [(11)C]cocaine binding potential in caudate (53.8% [13.8%]; 95% CI, 43.9% to 63.6%; P < .001), putamen (47.2% [11.4%]; 95% CI, 39.1% to 55.4%; P < .001), and nucleus accumbens (39.3% [10%]; 95% CI, 30% to 49%; P = .001), reflecting occupancy of dopamine transporters.
CONCLUSIONS: In this pilot study, modafinil blocked dopamine transporters and increased dopamine in the human brain (including the nucleus accumbens).
Because drugs that increase dopamine in the nucleus accumbens have the potential for abuse, and considering the increasing use of modafinil, these results highlight the need for heightened awareness for potential abuse of and dependence on modafinil in vulnerable populations.
A few notes on Ashwagandha, which I have been trying for the past few days. As far as my mood and clarity of thinking, its the best stuff I have tried yet.
I have 575 mgs tablets, a whole one during the day tends to make me a little too calm, but half a tab takes the edge off any stress or anxiety and lets me think clearly, and lasts 8 or 9 hours. It does seem to magnify the effects of alcohol, so be careful…
Yes piracetam potentiates many CNS-active substances. It is highly advisable to not drink in excess when taking it. You will get drunk faster and harder and this can turn into a bad thing if you are not prepared to handle blacking out…