I did the keto diet (strict under 20g carbs a day) for 3 months and lost 1.5 pounds. This was before TRT. Id be willing to give it another shot.
Ive been at it for about 2.5 weeks and have lost 11 pounds since I started. Just shredded. Iām hoping it balances out soon. I dont want to get back to my high school weight.
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God damn dude. What macros are you shooting for?
No macaroni for me
Lol sorry my brother. I dont know nuthin bout no macros. I aint measuring anything. Aint countin nuthin. Dropped the carbs and eating fibrous vegatables and meat and fat.
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Ok, I kind of figured but when someone says keto I assume they are measuring something, usually carbohydrate grams. So you just focus on veggie, meat, and fats and keep the rest out? Are you including PUFA like avocado or relying solely on animal and nut fat?
I ate my first avocado this morning with my morning eggs. I loathe avocados. Put enough sriracha sauce on it and its edible lol. And butter. I feel like I have a buzz all day I feel so good
Thats amazing that you feel so great dude. Dont forget the salt on those avocados. So aside from avo and eggs cooked in butter, what else are you eating today? And so help me christ if you say bunless McDonalds for lunch with a couple of atkins bars I quit.
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Had bulletproof coffee first thing before home gym workout. Post workout eggs with avocados. Snacking on peanuts. Had ground turkey for lunch. Not sure whats going down for dinner tonight. Lots of bacon. Grilled chicken for dinner maybe.
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Maybe Iāll do bacon wrapped chicken for dinner tonight!
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If you had to guess how many calories are you downing?
I would be totally guessing if I gave you a number
I need to get there. Though today someone called me a body builder and last week my barber asked if me if I was juicing. So I guess I finally look like I lift.
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I did the keto diet for about 9 weeks. After a while youāll start missing the glycogen needed for pushing hard in the gym. And youāll notice that you will start to lose some muscle mass as wellā¦
Keep an eye on the cholesterol too guys. Mine more than tripled on keto. Try to keep the sat fats to a minimum and eat poly unsats and good omega 3 and 6 as much as possibleā¦
Itās a great tool to have in your box for cutting fat really quickā¦I would NOT advise it as a long term lifestyleā¦
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As a matter of fact, you guys should read back in the earlier stages of this threadā¦(or maybe it was the AI Preachers thread?), where @physioLojik and I were going back and forth about keto. I was for and he was againstā¦he opened my eyes a bitā¦I found out the hard way that he was right lolā¦
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Trust me Iām still learning and in no way am I saying that this is the only way. Iām just trying to deal with my doctors orders. I have seen lots of info dismissing the dogma surrounding cholesterol. That triglycerides are more important than cholesterol blah blah blah. I hear what your saying about the carbs for excercise and Iām already seeing content about what they call ātargeted ketoā. It essentially uses high absorption dextrose pre workout to fuel the workout and then straight back into ketosis after. Ive watched videos of guys checking their blood and verifying ketosis within a few hours
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Yeah thereās a lot of back and forth on both sides of the fence. I watched mine on blood tests and it scared the shit out of me. I have a family history of heart disease, so I didnāt want to take the chanceā¦
It wasnāt so much that cholesterol was really high, which it wasā¦but it was that the LDL was through the roof, and the HDL/LDL ratio was way off. Itās hard to get enough to eat in fats to stay in ketosis without eating a high degree of saturated fatsā¦
I plan on using it anytime I go into a cut, donāt get me wrong. Iām not completely against it. It does work and very wellā¦
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I am genetically predisposed to not handle saturated fat very well. It may be why Ive not had much success with this style of eating. If I were to try it again I would have to focus on MUFAs and PUFAs.
Luckily these genetic traits do NOT put me at a greater risk for type 2 diabetes.
FTO rs17817449(G;G) Saturated fat may have a negative effect on blood glucose and insulin levels The FTO gene is called fat mass and obesity-associated protein because it is the gene that is the major genetic risk factor for obesity. This particular genotype, rs17817449(G;G), is associated with a 1.7-fold increased obesity risk in individuals with this genotype. Other gene polymorphisms in the , particularly in the context of a . Polyunsaturated fats are found in foods like fatty fish such as salmon, herring, as well as nuts. Saturated fat is found in fatty beef, pork, butter, cheese, and other dairy products. Individuals that are at high risk of obesity due to FTO polymorphisms may particularly benefit by having a higher polyunsaturated fat intake and a lower saturated fat intake.
FTO rs1121980(T;T) 2.76-fold increased risk for obesity particularly with saturated fat. The FTO gene is called fat mass and obesity-associated protein because it is the gene that is the
major genetic risk factor for obesity. This particular polymorphism is one of many in this gene that influences genetic obesity risk. This genotype, rs1121980(T;T), is associated with a 2.76-fold increased , particularly in the context of a Polyunsaturated fats are found in foods like fatty fish such as salmon, herring, and polyunsaturated fat is also found in nuts. Saturated fat is found in fatty beef, pork, butter, cheese, and other dairy products. This suggests that individuals that are at high risk of obesity due to FTO polymorphisms, such as this one, may benefit by having a higher polyunsaturated fat intake to saturated fat ratio.
PPAR gamma rs1801282(C;G) Abnormal fat metabolism Peroxisome proliferator-activated receptor gamma (PPAR-Ī³ or PPARG) is a master regulator of fatty acid storage and glucose metabolism. The genes activated by PPARG stimulate lipid uptake and adipogenesis by fat cells. It increases both lipid metabolism and adipogenesis in fat tissue. It increases insulin sensitivity in muscle tissue and increases gluconeogenesis in the liver. PPARG is mostly found in adipose tissue, colon and immune cells called macrophages. You may have an abnormal fat metabolism. When people with the genotype (C,G) or (G,G) have a low polyunsaturated and monounsaturated fat intake and a high saturated fat intake then their obesity and type 2 diabetes risks are dramatically increased. But when there is a higher mono- and poly-unsaturated intake and a lower saturated fat intake their obesity and type 2 diabetes risk is
normal. Polyunsaturated fatty acids activate the PPAR-gamma gene. Monounsaturated fat is found in foods such as avocado, olive oil, and nuts. Polyunsaturated fats are found in foods like fatty fish such as salmon, herring and polyunsaturated fat is also found in nuts. Saturated fat is found in fatty beef, pork, butter, cheese, and other dairy products.
Individuals with the genotypes (C,G) or (G,G) may want to make sure their dietary fat is higher in
mono- and poly-unsaturated fat and lower in saturated fat.Pterostilbene is a plant compound present in blueberries, cranberries, and almonds
ACE rs4343(G;G) Increased risk of elevated blood pressure and impaired glucose metabolism in response to a high saturated fat diet The ACE gene codes for the angiotensin-converting enzyme, a regulatory enzyme in the reninangiotensin system, which plays an important role in the regulation of blood pressure. There is a common insertion/deletion of 287 base pairs in this gene, for which rs4343 serves as a proxy (surrogate marker). Those with the A allele have the insertion and those with the G allele have the deletion. The deletion (G) has been shown to result in higher levels of the ACE enzyme in the serum. This genotype, rs4343(G;G) is associated with an increased risk of elevated blood pressure and impaired glucose metabolism in response to a high saturated fat diet.
FADS1-1 rs174550(C;C) Increased inflammation in conjunction with a high
linoleic acid (omega-6) diet Fatty acid desaturase 1 (FADS1) is an enzyme involved in the metabolism of polyunsaturated fatty acids (PUFAs). The essential fatty acids, linoleic acid (LA, omega-6) and alpha-linolenic acid (ALA, omega-3) compete for the FADS1 enzyme. The omega-6 pathway leads to arachidonic acid (AA), a precursor of pro-inflammatory compounds, while the omega-3 pathway leads to eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA) which tend to be anti-inflammatory. This genotype, rs174550(C;C), has been associated with increased inflammation in conjunction with a high linoleic acid (omega-6) diet.
Thatās good info to have right there!!
Question, if Iām not getting too personal hereā¦
What is your blood type? Iām guessing A or B and not O. Curious to compare to some other research I have done regarding genetics and the effects of diet vs genetic profiling.