I eat fairly low carb; I feel better and fat melts off. So, a lot of chicken and dairy products, sometimes beef… supplement with protein shakes and bars when necessary.
Ironically, I do not have any reasonable TRT lipid blood work because none of my previous providers assessed lipids. I just began getting them (at my own expense) because I knew I wanted to try a cycle and figured I should be looking at kidney/liver/lipids to do it responsibly.
I’d recommend after your blast that you drop to 200mg/wk testosterone ONLY. This should give you high normal total testosterone tested in the trough.
I don’t know how long you like to blast, but I would keep blasts to 8 weeks and cruise at least 8 weeks. Test bloods 7 weeks into the cruise to see if bloods are close to normal. If not, cruise 4 more weeks. Test again.
I don’t have a preferred blast duration yet since this is my first one, so I’m hoping I can get this sorted and find a reasonably healthy way to proceed.
I will heed this advice and post updated labs once everything has had time to clear my system.
Agreed. Interesting that TRT alone seemed to pretty dramatically impact cholesterol for the worse.
The main change from a dietary perspective was a move away from carbohydrate consumption. I went from probably 400g of carbs daily (I never really counted, but in an undisciplined state, I eat a lot of pizza and rice) to <150g daily now.
I agree. I wouldn’t go to that extreme with the not training before, but Imo if someone is developed enough to use AAS, they aren’t getting that strength increase. Going from 225 to 250 lbs, sure, but imo, most shouldn’t be using if they bench 225 for 1.
I guess with blast and cruise I went from 185 lb bench to 405 lbs in under a year, but before that 185 max, I did 385, then messed up my shoulder in a way that just destroyed my strength.
So, I dwelled on this last night and I have a strong suspicion that deca is actually the main culprit in hurting my lipids. Considering my pre-anavar bloodwork showed lipids trending in the wrong direction and deca was a variable I had already introduced, I’m wondering if that was really the main source of the problem and anavar (a known factor in crashing lipids) just tipped things over from bad to worse. It will be interesting to see cruise bloodwork that would potentially validate this. I’ll update after my next round of labs so that data point will be available for others.