0.5ed letro is WAAAAYYYYY on the high side.
I am trying to find a chart I came across a while ago to better explain, but it essentially listed letro as the most powerful AI iirc. You would have an E2 armageddon.
[quote]33 Big Blinds wrote:
0.5ed letro is WAAAAYYYYY on the high side.
I am trying to find a chart I came across a while ago to better explain, but it essentially listed letro as the most powerful AI iirc. You would have an E2 armageddon.[/quote]
True but the funny thing is I’ve been up to 3.25mg ED with letro (to reverse gyno) and I had no low E2 symptoms compared to my adex use where if I take 0.5mg ED instead of 0.25mg my joints are on the verge of snapping.
Interesting.
SB
Singh - how long did you run at that dose (3.25 mg ED) - ?
Also, how bad was your gyno, and did you get a full reversal of said gyno?
Just curious - thanks brother!
[quote]heg842 wrote:
Singh - how long did you run at that dose (3.25 mg ED) - ?
Also, how bad was your gyno, and did you get a full reversal of said gyno?
Just curious - thanks brother![/quote]
The gyno was fairly pronounced, noticeable by me definetly but ‘puffy’ to the average joe.
Obviously puffiness is not gyno as you know, but upon applying pressure to the areola I could feel lumps (breast tissue) and it was painful for the first time ever.
I ran it at that dose for roughly a week and did not see an improvement, my letro was pharm-grade. Either my E2 was sky-high and even 3.25mg was not enough to bring it down or something else was up. My best guess would be prolactin (although there was no lactation).
It makes me curious however as I have read that 0.25mg of letro can block up to 97% of oestrogen, some food for thought there.
No probs, happy to help.
SB
Your pituitary will stop producing LH when you are injecting HCG/Ovidrel but it’s a bit more complex than simply a yes/no answer to it being completely counter productive. In healthy males post AAS cycle LH levels bounce back relatively quickly on their own even with no PCT compared to the time it takes for the testes to start producing testosterone. The main hurdle in HTPA recovery is testicular desensitisation to LH due to them being out of use for so long. The longer you are on AAS ->the more your balls will atrophy → the more desensitised to LH they will become → the longer it takes for the LH in your body to actually be effective for test production. So using HCG on cycle will keep your testes ready to produce endogenous test as soon as your serum testosterone levels drop to the the normal range with the help of clomid/nolva to kickstart to the pituitary.
This is not to say that HCG/Ovidrel is completely counter productive during PCT either. With the help of clomid/nolva, as soon as HCG levels drop off, your natural LH production actually kicks in very quickly and your natural test starts flowing again. The clomid/nolva is still ‘waking up’ the pituitary even though it might not be gushing out LH, but it makes sense that the clomid/nolva would be used more effectively if you were churning out LH while taking it!
I have seen those other forums and they are “duh”. They think a 12 week oral only with no pct is a great idea. And they agree with whatever you say. Fuck you could say " I like to inject only on sunny days at 2 because I turn my ass to the sun and the uv rays help it enter smoother." than 6 people reply how they tried it and loved it.
Here you don’t get babied. You get real answers and told when your wrong. Cause that’s what helps keep people safe. Is the real information
[quote]Slabbie wrote:
Your pituitary will stop producing LH when you are injecting HCG/Ovidrel but it’s a bit more complex than simply a yes/no answer to it being completely counter productive. In healthy males post AAS cycle LH levels bounce back relatively quickly on their own even with no PCT compared to the time it takes for the testes to start producing testosterone. The main hurdle in HTPA recovery is testicular desensitisation to LH due to them being out of use for so long. The longer you are on AAS ->the more your balls will atrophy → the more desensitised to LH they will become → the longer it takes for the LH in your body to actually be effective for test production. So using HCG on cycle will keep your testes ready to produce endogenous test as soon as your serum testosterone levels drop to the the normal range with the help of clomid/nolva to kickstart to the pituitary.
This is not to say that HCG/Ovidrel is completely counter productive during PCT either. With the help of clomid/nolva, as soon as HCG levels drop off, your natural LH production actually kicks in very quickly and your natural test starts flowing again. The clomid/nolva is still ‘waking up’ the pituitary even though it might not be gushing out LH, but it makes sense that the clomid/nolva would be used more effectively if you were churning out LH while taking it![/quote]
Now you are just making shit up…You say the testes are the last to reccover after a cycle? In what fucking universe? I can’t even begin to count how many guys have come into the TRT forum with Low T after doing a cycle, and when they get blood work done their FSH/LH are in the shitter…as soon as they run a proper PCT, these values rise and their testosterone rises with it. You have absolutely no clue what you are talking about.