Got my labs, I didn't specify that I wanted to see my free-test, oh well.
Anyway, my prolactin level is 16 ng/ml. It seems high. What kind of effect would this have on me?
Also, my LH is 7.7 Miu/ml which is also at the higher end. Is that a good thing?
Higher LH is a good thing IMHO. Don’t think you should worry about that. If you’re early into a cycle right now, then that should make sense. If not on, then I wouldn’t worry about it either.
As far as prolactin levels go, I’m not really sure what effects a higher level would have on you without reviewing a bit about it first. 'Bout the only thing I can think of off the top of my head is that your refractory period might be on the high/long side after doing the dirty boogie. 
I imagine a low dose of dostinex would bring that into optimal alignment. But I’m not really qualified to say, and I’m shooting from the hip.
Thanks. I read more into it and it appears dostinex is what would be prescribed. I did find a bean called the “velvet bean” which when sprouted contains EXTREMELY high levels of l-dopa, a carboxylated version of dopamine, which is the antagonist of prolactin! This might be very useful for individuals experiencing problems with DHT derived AAS.
[quote]Diana Bolann wrote:
Got my labs, I didn’t specify that I wanted to see my free-test, oh well.
Anyway, my prolactin level is 16 ng/ml. It seems high. What kind of effect would this have on me?
Also, my LH is 7.7 Miu/ml which is also at the higher end. Is that a good thing?[/quote]
Context:
At my lab, both your Prolactin and LH are in the normal range. Normal is normal; the hour to hour variation and lab sensitivities don’t allow us to pathologize normal values.
Do NOT take dostinex or dopa. Remember: you have hypertension and are taking metoprolol. Do any of our friends posting here know the potential interactions? Why treat something that is normal? How many doses does it take to produce valvular disease? I don’t know and I don’t want you to find out first hand.
The LH is instructive: if you have been taking a non-aromatizing steroid, or sufficient AI, the LH would not be suppressed by HRT; estrogen and DHT are the stronger suppressants of GnRH and LH.
[quote]DrSkeptix wrote:
Can you interpret your third point for me? I don’t understand what you mean.
[quote]beebuddy wrote:
DrSkeptix wrote:
Can you interpret your third point for me? I don’t understand what you mean.[/quote]
In terms of suppression of LH and FSH secretion from the Pituitary, estradiol is stronger suppressant than DHT, DHT stronger than T. Estrogen also suppresses GnRH secretion from hypothalamus, so LH and FSH can be suppressed by estrogen at two levels.
A thought experiment:
Now imagine that someone-lets call him DB–takes an non-aromatized anabolic which is weakly androgenic; say, Anavar (oxandrolone) T may be lowered some, DHT may be lowered some, and estrogen will be very low, so LH may be normal or high normal (“unsuppressed.”) This experiment has not been done, to my knowledge, so other responses might follow.
Scenario #2: DB takes an AI, with or without an strong anabolic/weak androgen. SImilarly, LH may be normal or high normal. (The AI only experiment has been done in hypogonadal men, and yes, it happens this way.)
Notice I use the word “may.” To my knowledge, this type of experiment has not been done. The choice of which AI and AAS used may influence the results.