Many athletes have an increased thickness of the left ventricle (I’ve seen reports of supposedly NATURAL athletes (mostly ultra endurance and powerlifting athletes) having LVWT of up to 16-19mm, that being said the majority of individuals, even athletes will fall between the 10-12mm
The cause of athletic cardiomegaly is due to prolonged increase of cardiac output, thus the chambers of the left ventricle enlarge to compensate for frequently increased cardiac output. There’s two types of LVH, concentric and eccentric. I find the heart to be a fascinating organ, so if you wish for me to explain all this jazz in detail shoot me an email (in my bio) and I’ll be more than happy to talk about it.
Anabolic steroids tend to cause LVH through various mechanisms, and tend to exacerbate the LV hypertrophic response in response to intense weight training/ athletic activity. That being said, androgens appear to have a direct effect on the myocardium, cardiac myocytes contain androgen receptors, many actions of AAS are mediated by AR binding thus AR binding to cardiac myocytes = cardiac hypertrophy. The extent of the LVH will depend on lifestyle, genetics (someone with familial inhereted HCM will have a fucking huge heart on AAS lol, with equally huge arrythmogenic potential). Other mechanisms are, but not limited to, high blood pressure via excess sodium retention and other mechanisms caused by AAS, beta adrenergic receptor upregulation, oxidive stress and increased free radicals circulating in the body, (tachycardia induced cardiomyopathy for those extremely sensitive to this effect), individual amount of AR in cardiomyopathy and indirectly a shitty lifestyle, diet and whatnot will correlate with how big you’re heart gets on AAS.
Various supplements can help with oxidive stress and antioxidant dysfunction induced by AAS, I like Curcumin, coq-10, grape seed extract, garlic (I love garlic due to the taste lol), hawthorn berry and more, however just because you’re taking these supps doesn’t mean you’re risk free).
What’s the issue with an enlarged heart? If the left ventricular walls themselves become too thick, the amount of blood the heart can pump becomes impaired, or if it becomes too stretched (weak) the same occurs. LVH is arrythmogenic and increases you’re risk for deadly arrythmias like v-fib. What’s you’re resting HR? Most who have athletic cardiomyopathy will exhibit bradycardia. Do you have symptoms such as arrythmias, chest pain, synocope upon exercise, shortness of breath, swelling of the legs etc. The individuals (natural) I know who had HCM ( not family or related to me in any way ) only found out after they went into cardiac arrest, their ankles started swelling up etc. You need to get you’re cardiac function and output tested, a halter moniter might be good to see if you have any arrythmias throughout the day.
How many blasts have you run? What’s the highest dose you’ve used?
AAS aren’t harmless, it irritates me when people completely gloss over the potential cardiac risks. Be informed, stay smart, be safe.
Post could’ve been longer but I don’t want it being so ridiculously long you won’t read it lol
In my non professional opinion LV enlargement is only clinically significant if its associated with cardiac dysfunction. Other things that can cause LVH are (but not limited to) various cardiotoxic drugs (amphetamines, doxorubicin, cocaine), excess alcohol consumption, being a fat person, diabetes, being PREGNANT (although I don’t think you have to worry about that lol) and more