Your debate style is to essentially flood. Any claim that is made that contradicts yours is met with rapid-fire posting and appeals to abstract authority instead of addressing the claim with reason.
I’m not going to ignore the study you provided. I will read it. I’ll send it to Jordan as well and get his feedback and reply here. Post the statement you would like me to share with all of them and I’ll post their replies for you.
In case you or anyone else is interested in the technical name for your style of rhetorical debate, it’s known as “gish gallop.” It’s an indication the debater isn’t willing to engage in reasoned discourse.
I absolutely am. Again, for the 5th time, your statement was you are sensitive to E2 because it causes gyno and you need an AI.
It’s false. Even the study you provided displays several reasons for this. First and foremost you have to be genetically predisposed to get it in the first place. It’s one study. I can send you 50 that completely contradict it.
Again, if E2 was the cause of gyno, all these physicians would be prescribing an AI. They used to and stopped because they know better now. Tens of thousands of men. You can ask them yourself and get it from the horse’s mouth and remove me from the equation. I don’t know what more I can be providing here. All you’re providing, aside from a single study, is that you don’t like how I debate. It’s weak.
If this was true, and we know that Arimidex lowers estradiol, why did I develop gyno with an E2 of 12? Why did the gyno shrink with an E2 of 60. This is but ONE example, that I can demonstrate with lab work, that completely contradicts your claim and I’m but one single example.
Please explain this to me like I’m 4.
No, that’s not my statement. That is your reframing of my statement.
No, it doesn’t. It fully supports my central claim.
This is an error in logic and appeal to authority. Physicians also used to lobotomize people.
Whether I like it doesn’t make a difference. It’s that it prevents reasoned discourse.
Potentially because, as you’ve said yourself, serum levels of E2 are meaningless and do not indicate tissue action. My claim isn’t based from my own experience. It’s based on reasoning and information gleaned from the most reputable clinics in the world.
You have the audacity to claim and appeal to authority and say this? Lol!
Let’s make one thing perfectly clear.
You’ve provided me with a SINGLE STUDY. I’ve provided you with significantly more evidence. I stated I would send your study to these physicians for review and I’ll review it myself. You have immediately dismissed all the evidence I’ve provided and stick to your one single study.
If this isn’t the very definition of confirmation bias, I don’t know what is.
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We have to go back to some source, don’t we? At least my authority isn’t an abstraction of doctors.
The study you linked has nothing whatsoever to do with my claim. You have yet to address the study I linked other than to say you would gather the opinions of doctors.
Your SOURCE is the 'the best physicians in the world". Who?
My SOURCES are also the best physicians in the world who I can put you in direct contact with TODAY so that you can speak to them DIRECTLY. Not to mention who will drown you in research and data as to why they changed their tune regarding AI use.
Who is more credible here?
I actually need some time to read the damn thing, as I said I would, and get back to you.
@highpull is this what you were talking about the other day? 
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This is just not about credibility, Danny. I see that you are very sensitive to being perceived as “credible.” That’s understandable given the reputational stake you have here. I am making but as single, simple claim.
There is no credibility issue. There is only evidence or a lack of evidence. I have posted a study that backs up my simple claim. You have not posted any evidence that refutes it.
Spoiler alert: I don’t give a rat’s ass about my reputation or being credible. This is a hobby for me. This has ZERO impact on my reality. If I really, truly cared about my ‘reputation’ I would be unbelievably ‘professional and polite and well-spoken’. I’m clearly not doing any of that here, am I?
You posted a study that stated estradiol can cause it. The very same study stated that estradiol required issues with growth hormone and IGF-1 debunking your claim. There is literature to suggest a multitude of factors that can cause gyno with estradiol not being the single cause as you were leading everyone to believe.
You’re also not posting any evidence to refute the claim I’ve made.
Read above comment.
Furthermore, other data indicates that estrogen promotes GH secretion and increases GH levels, stimulating the production of IGF-1, which synergizes with estrogen to induce ductal development.
So, estrogen promotes GH secretion which causes gyno… so why are there so many men on HGH therapy not getting gyno? Hmmm…
There are a multitude of factors. There is not one unifying factor. I know you don’t get it. Keep taking the AI.
Ok, since you tagged me in, I’ll play.
The problem is your levels are not stable and your body cannot handle the fluctuations with the massive dose of testosterone you are taking. I would take 2.5mg injected every six hours. Set your alarm clock if you sleep over six hours, or use 3.3mg every eight hours.
Seriously guys, take your testosterone and enjoy your life with the benefits. Don’t obsess over side effects, or you’ll have them. It’s ok if you’re tired today, or anxious, or you had a bad workout, or you don’t feel like sex two or three times today. Very few have trouble with estrogen.
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Estrogen effects on the breast might be the result of circulating estradiol levels or locally produced estrogens. Aromatase P450 catalyzes the conversion of the C19 steroids, androstenedione, testosterone, and 16−α−hydroxyandrostenedione to estrone, estradiol-17β, and estriol. As such, an overabundance of substrate or an increase in enzyme activity can increase estrogen concentrations and initiate the cascade to breast development in females and males. For example, in the more complete forms of androgen insensitivity syndromes in genetically male (XY) patients, excess androgen aromatizes into estrogen resulting in not only gynecomastia, but also a phenotypic female appearance.
@spinup how about asking the physician who just chimed in? Not a layman.
You’re avoiding addressing the evidence I am posting.